Erich Wanker
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Explore the profile of Erich Wanker including associated specialties, affiliations and a list of published articles.
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Articles
22
Citations
435
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Recent Articles
1.
Lorenz C, Zink A, Henke M, Staege S, Mlody B, Bunning M, et al.
Stem Cell Res
. 2022 Mar;
61:102742.
PMID: 35279592
We report the generation of four human iPSC lines (8993-A12, 8993-B12, 8993-C11, and 8993-D7) from fibroblasts of four patients affected by maternally inherited Leigh syndrome (MILS) carrying homoplasmic mutations m.8993T ...
2.
Zink A, Conrad J, Telugu N, Diecke S, Heinz A, Wanker E, et al.
Front Cell Dev Biol
. 2020 Nov;
8:590540.
PMID: 33224955
Excessive ethanol exposure can cause mitochondrial and cellular toxicity. In order to discover potential counteracting interventions, it is essential to develop assays capable of capturing the consequences of ethanol exposure...
3.
Poh J, Ponsford A, Boyd J, Woodsmith J, Stelzl U, Wanker E, et al.
Redox Biol
. 2020 Sep;
37:101686.
PMID: 32911434
NRF2 (NFE2L2) is a cytoprotective transcription factor associated with >60 human diseases, adverse drug reactions and therapeutic resistance. To provide insight into the complex regulation of NRF2 responses, 1962 predicted...
4.
Schilling J, Broemer M, Atanassov I, Duernberger Y, Vorberg I, Dieterich C, et al.
J Mol Biol
. 2019 Feb;
431(9):1869-1877.
PMID: 30711541
Huntington's disease (HD) is caused by an expanded CAG repeat in the huntingtin (HTT) gene, translating into an elongated polyglutamine stretch. In addition to the neurotoxic mutant HTT protein, the...
5.
Arnoux I, Willam M, Griesche N, Krummeich J, Watari H, Offermann N, et al.
Elife
. 2018 Sep;
7.
PMID: 30179155
Catching primal functional changes in early, 'very far from disease onset' (VFDO) stages of Huntington's disease is likely to be the key to a successful therapy. Focusing on VFDO stages,...
6.
Matthes F, Hettich M, Schilling J, Flores-Dominguez D, Blank N, Wiglenda T, et al.
Cell Death Discov
. 2018 Mar;
4:4.
PMID: 29531801
Alzheimer's disease (AD) is characterized by two neuropathological hallmarks: senile plaques, which are composed of amyloid-β (Aβ) peptides, and neurofibrillary tangles, which are composed of hyperphosphorylated tau protein. Aβ peptides...
7.
Matthes F, Massari S, Bochicchio A, Schorpp K, Schilling J, Weber S, et al.
ACS Chem Neurosci
. 2018 Mar;
9(6):1399-1408.
PMID: 29506378
Expanded CAG trinucleotide repeats in Huntington's disease (HD) are causative for neurotoxicity. The mutant CAG repeat RNA encodes neurotoxic polyglutamine proteins and can lead to a toxic gain of function...
8.
Andrich K, Hegenbart U, Kimmich C, Kedia N, Bergen 3rd H, Schonland S, et al.
J Biol Chem
. 2016 Dec;
292(6):2328-2344.
PMID: 28031465
Intervention into amyloid deposition with anti-amyloid agents like the polyphenol epigallocatechin-3-gallate (EGCG) is emerging as an experimental secondary treatment strategy in systemic light chain amyloidosis (AL). In both AL and...
9.
Mazza A, Klockmeier K, Wanker E, Sharan R
Bioinformatics
. 2016 Dec;
32(24):3855.
PMID: 27993841
No abstract available.
10.
Mazza A, Klockmeier K, Wanker E, Sharan R
Bioinformatics
. 2016 Jun;
32(12):i271-i277.
PMID: 27307626
Motivation: Unraveling the molecular mechanisms that underlie disease calls for methods that go beyond the identification of single causal genes to inferring larger protein assemblies that take part in the...