Elinor Ng Eaton
Overview
Explore the profile of Elinor Ng Eaton including associated specialties, affiliations and a list of published articles.
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Snapshot
Articles
11
Citations
6753
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0
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Recent Articles
1.
Phadnis V, Snider J, Varadharajan V, Ramachandiran I, Deik A, Lai Z, et al.
Cell Rep
. 2023 Sep;
42(9):113023.
PMID: 37691145
Ferroptosis is a form of regulated cell death with roles in degenerative diseases and cancer. Excessive iron-catalyzed peroxidation of membrane phospholipids, especially those containing the polyunsaturated fatty acid arachidonic acid...
2.
Lambert A, Fiore C, Chutake Y, Verhaar E, Strasser P, Chen M, et al.
Dev Cell
. 2022 Dec;
57(24):2714-2730.e8.
PMID: 36538894
Cancer stem cells (CSCs) may serve as the cellular seeds of tumor recurrence and metastasis, and they can be generated via epithelial-mesenchymal transitions (EMTs). Isolating pure populations of CSCs is...
3.
Dongre A, Rashidian M, Eaton E, Reinhardt F, Thiru P, Zagorulya M, et al.
Cancer Discov
. 2020 Dec;
11(5):1286-1305.
PMID: 33328216
The epithelial-to-mesenchymal transition, which conveys epithelial (E) carcinoma cells to quasi-mesenchymal (qM) states, enables them to metastasize and acquire resistance to certain treatments. Murine tumors composed of qM mammary carcinoma...
4.
Kroger C, Afeyan A, Mraz J, Eaton E, Reinhardt F, Khodor Y, et al.
Proc Natl Acad Sci U S A
. 2019 Mar;
116(15):7353-7362.
PMID: 30910979
Carcinoma cells residing in an intermediate phenotypic state along the epithelial-mesenchymal (E-M) spectrum are associated with malignant phenotypes, such as invasiveness, tumor-initiating ability, and metastatic dissemination. Using the recently described...
5.
Lu H, Clauser K, Tam W, Frose J, Ye X, Eaton E, et al.
Nat Cell Biol
. 2015 Nov;
17(12):1607.
PMID: 26612575
No abstract available.
6.
Ye X, Tam W, Shibue T, Kaygusuz Y, Reinhardt F, Eaton E, et al.
Nature
. 2015 Sep;
525(7568):256-60.
PMID: 26331542
Tumour-initiating cells (TICs) are responsible for metastatic dissemination and clinical relapse in a variety of cancers. Analogies between TICs and normal tissue stem cells have led to the proposal that...
7.
Lu H, Clauser K, Tam W, Frose J, Ye X, Eaton E, et al.
Nat Cell Biol
. 2014 Oct;
16(11):1105-17.
PMID: 25266422
The cell-biological program termed the epithelial-mesenchymal transition (EMT) confers on cancer cells mesenchymal traits and an ability to enter the cancer stem cell (CSC) state. However, the interactions between CSCs...
8.
Scheel C, Eaton E, Li S, Chaffer C, Reinhardt F, Kah K, et al.
Cell
. 2011 Jun;
145(6):926-40.
PMID: 21663795
The epithelial-mesenchymal transition (EMT) has been associated with the acquisition of motility, invasiveness, and self-renewal traits. During both normal development and tumor pathogenesis, this change in cell phenotype is induced...
9.
Kojima Y, Acar A, Eaton E, Mellody K, Scheel C, Ben-Porath I, et al.
Proc Natl Acad Sci U S A
. 2010 Nov;
107(46):20009-14.
PMID: 21041659
Much interest is currently focused on the emerging role of tumor-stroma interactions essential for supporting tumor progression. Carcinoma-associated fibroblasts (CAFs), frequently present in the stroma of human breast carcinomas, include...
10.
Mani S, Guo W, Liao M, Eaton E, Ayyanan A, Zhou A, et al.
Cell
. 2008 May;
133(4):704-15.
PMID: 18485877
The epithelial-mesenchymal transition (EMT) is a key developmental program that is often activated during cancer invasion and metastasis. We here report that the induction of an EMT in immortalized human...