David R Stanford
Overview
Explore the profile of David R Stanford including associated specialties, affiliations and a list of published articles.
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14
Citations
306
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Recent Articles
1.
Tooley K, Chucair-Elliott A, Ocanas S, Machalinski A, Pham K, Hoolehan W, et al.
Epigenetics Chromatin
. 2023 Nov;
16(1):45.
PMID: 37953264
Background: Cellular identity is determined partly by cell type-specific epigenomic profiles that regulate gene expression. In neuroscience, there is a pressing need to isolate and characterize the epigenomes of specific...
2.
Tooley K, Chucair-Elliott A, Ocanas S, Machalinski A, Pham K, Stanford D, et al.
bioRxiv
. 2023 Jun;
PMID: 37333391
Background: Cellular identity is determined partly by cell type-specific epigenomic profiles that regulate gene expression. In neuroscience, there is a pressing need to isolate and characterize the epigenomes of specific...
3.
Ocanas S, Ansere V, Tooley K, Hadad N, Chucair-Elliott A, Stanford D, et al.
Mol Neurobiol
. 2022 May;
59(8):4669-4702.
PMID: 35589920
Common neurological disorders, like Alzheimer's disease (AD), multiple sclerosis (MS), and autism, display profound sex differences in prevalence and clinical presentation. However, sex differences in the brain with health and...
4.
Chucair-Elliott A, Ocanas S, Stanford D, Ansere V, Buettner K, Porter H, et al.
Commun Biol
. 2020 Nov;
3(1):693.
PMID: 33214681
Epigenetic regulation of gene expression occurs in a cell type-specific manner. Current cell-type specific neuroepigenetic studies rely on cell sorting methods that can alter cell phenotype and introduce potential confounds....
5.
Hadad N, Masser D, Blanco-Berdugo L, Stanford D, Freeman W
Epigenetics Chromatin
. 2019 Oct;
12(1):58.
PMID: 31594536
Background: Alterations to cellular and molecular programs with brain aging result in cognitive impairment and susceptibility to neurodegenerative disease. Changes in DNA methylation patterns, an epigenetic modification required for various...
6.
Chucair-Elliott A, Ocanas S, Stanford D, Hadad N, Wronowski B, Otalora L, et al.
Geroscience
. 2019 Sep;
41(5):691-708.
PMID: 31493147
The systemic delivery of tamoxifen (Tam) to activate inducible CreERT2-loxP transgenic mouse systems is now widely used in neuroscience studies. This critical technological advancement allows temporal control of DNA-cre recombination,...
7.
Imperio C, McFalls A, Hadad N, Blanco-Berdugo L, Masser D, Colechio E, et al.
Neuropharmacology
. 2018 Jul;
139:26-40.
PMID: 29964093
Environmental factors profoundly affect the addictive potential of drugs of abuse and may also modulate the neuro-anatomical/neuro-chemical impacts of uncontrolled drug use and relapse propensity. This study examined the impact...
8.
Hadad N, Unnikrishnan A, Jackson J, Masser D, Otalora L, Stanford D, et al.
Neurobiol Aging
. 2018 Apr;
67:53-66.
PMID: 29631215
Brain aging is marked by cognitive decline and susceptibility to neurodegeneration. Calorie restriction (CR) increases neurogenesis, improves memory function, and protects from age-associated neurological disorders. Epigenetic mechanisms, including DNA methylation,...
9.
Masser D, Hadad N, Porter H, Stout M, Unnikrishnan A, Stanford D, et al.
Geroscience
. 2018 Jan;
40(1):11-29.
PMID: 29327208
As geroscience research extends into the role of epigenetics in aging and age-related disease, researchers are being confronted with unfamiliar molecular techniques and data analysis methods that can be difficult...
10.
Masser D, Hadad N, Porter H, Mangold C, Unnikrishnan A, Ford M, et al.
Aging Cell
. 2017 Sep;
16(6):1342-1352.
PMID: 28948711
DNA methylation is a central regulator of genome function, and altered methylation patterns are indicative of biological aging and mortality. Age-related cellular, biochemical, and molecular changes in the hippocampus lead...