David M Gilbert
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Explore the profile of David M Gilbert including associated specialties, affiliations and a list of published articles.
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8941
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Recent Articles
1.
Hartley G, Okhovat M, Hoyt S, Fuller E, Pauloski N, Alexandre N, et al.
Cell Genom
. 2025 Mar;
:100808.
PMID: 40088887
Great apes have maintained a stable karyotype with few large-scale rearrangements; in contrast, gibbons have undergone a high rate of chromosomal rearrangements coincident with rapid centromere turnover. Here, we characterize...
2.
Turner J, Hinojosa-Gonzalez L, Sasaki T, Uchino S, Vouzas A, Soto M, et al.
bioRxiv
. 2025 Feb;
PMID: 39990485
Eukaryotic genomes replicate in a defined temporal order called the replication timing (RT) program. RT is developmentally regulated with potential to drive cell fate transitions, but mechanisms controlling RT remain...
3.
Vouzas A, Sasaki T, Rivera-Mulia J, Turner J, Brown A, Alexander K, et al.
bioRxiv
. 2025 Feb;
PMID: 39975371
DNA replication timing (RT) is correlated with transcription during cell fate changes but there are many exceptions and our understanding of this relationship suffers from a paucity of reductionist approaches....
4.
Dekker J, Oksuz B, Zhang Y, Wang Y, Minsk M, Kuang S, et al.
bioRxiv
. 2024 Nov;
PMID: 39484446
The dynamic three-dimensional (3D) organization of the human genome (the "4D Nucleome") is closely linked to genome function. Here, we integrate a wide variety of genomic data generated by the...
5.
Kumar P, Gholamalamdari O, Zhang Y, Zhang L, Vertii A, van Schaik T, et al.
Commun Biol
. 2024 Sep;
7(1):1135.
PMID: 39271748
Genome differential positioning within interphase nuclei remains poorly explored. We extended and validated Tyramide Signal Amplification (TSA)-seq to map genomic regions near nucleoli and pericentric heterochromatin in four human cell...
6.
Hartley G, Okhovat M, Hoyt S, Fuller E, Pauloski N, Alexandre N, et al.
bioRxiv
. 2024 Sep;
PMID: 39257810
Great apes have maintained a stable karyotype with few large-scale rearrangements; in contrast, gibbons have undergone a high rate of chromosomal rearrangements coincident with rapid centromere turnover. Here we characterize...
7.
Anderson C, Talmane L, Luft J, Connelly J, Nicholson M, Verburg J, et al.
Nature
. 2024 Jun;
630(8017):744-751.
PMID: 38867042
DNA base damage is a major source of oncogenic mutations. Such damage can produce strand-phased mutation patterns and multiallelic variation through the process of lesion segregation. Here we exploited these...
8.
Gholamalamdari O, van Schaik T, Wang Y, Kumar P, Zhang L, Zhang Y, et al.
bioRxiv
. 2024 May;
PMID: 38712201
Models of nuclear genome organization often propose a binary division into active versus inactive compartments yet typically overlook nuclear bodies. Here we integrated analysis of sequencing and image-based data to...
9.
Corazzi L, Ionasz V, Andrejev S, Wang L, Vouzas A, Giaisi M, et al.
Nat Commun
. 2024 Apr;
15(1):3594.
PMID: 38678011
Recurrent DNA break clusters (RDCs) are replication-transcription collision hotspots; many are unique to neural progenitor cells. Through high-resolution replication sequencing and a capture-ligation assay in mouse neural progenitor cells experiencing...
10.
Hogan M, Holding M, Nystrom G, Colston T, Bartlett D, Mason A, et al.
Proc Natl Acad Sci U S A
. 2024 Apr;
121(16):e2313440121.
PMID: 38578985
Developmental phenotypic changes can evolve under selection imposed by age- and size-related ecological differences. Many of these changes occur through programmed alterations to gene expression patterns, but the molecular mechanisms...