David B Hood
Overview
Explore the profile of David B Hood including associated specialties, affiliations and a list of published articles.
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Articles
6
Citations
240
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0
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Recent Articles
1.
Hood D, Snyder K, Buckner T, Hurley B, Pitts K, Lopez L
Eur J Rheumatol
. 2016 Oct;
2(4):135-138.
PMID: 27708950
Objective: The routine measurement of IgA anticardiolipin (aCL) and IgA anti-β glycoprotein I (anti-β GPI) antibodies remain controversial despite several studies demonstrating an association with thromboembolic disease in patients with...
2.
Meredith E, Beattie K, Burgis R, Capparelli M, Chapo J, DiPietro L, et al.
J Med Chem
. 2010 Aug;
53(15):5422-38.
PMID: 20684592
The synthesis and biological evaluation of potent and selective PKD inhibitors are described herein. The compounds described in the present study selectively inhibit PKD among other putative HDAC kinases. The...
3.
Monovich L, Vega R, Meredith E, Miranda K, Rao C, Capparelli M, et al.
FEBS Lett
. 2009 Dec;
584(3):631-7.
PMID: 20018189
Class IIa histone deacetylases (HDACs) repress genes involved in pathological cardiac hypertrophy. The anti-hypertrophic action of class IIa HDACs is overcome by signals that promote their phosphorylation-dependent nuclear export. Several...
4.
Monovich L, Koch K, Burgis R, Osimboni E, Mann T, Wall D, et al.
Biochim Biophys Acta
. 2009 May;
1789(5):422-31.
PMID: 19414071
Histone deacetylase 5 (HDAC5) represses expression of nuclear genes that promote cardiac hypertrophy. Agonism of a variety of G protein coupled receptors (GPCRs) triggers phosphorylation-dependent nuclear export of HDAC5 via...
5.
Bush E, Hood D, Papst P, Chapo J, Minobe W, Bristow M, et al.
J Biol Chem
. 2006 Sep;
281(44):33487-96.
PMID: 16950785
The calcium/calmodulin-dependent phosphatase calcineurin plays a central role in the control of cardiomyocyte hypertrophy in response to pathological stimuli. Although calcineurin is present at high levels in normal heart, its...
6.
Harrison B, Roberts C, Hood D, Sweeney M, Gould J, Bush E, et al.
Mol Cell Biol
. 2004 Dec;
24(24):10636-49.
PMID: 15572669
Diverse pathological insults trigger a cardiac remodeling process during which myocytes undergo hypertrophy, with consequent decline in cardiac function and eventual heart failure. Multiple transcriptional regulators of pathological cardiac hypertrophy...