D Modrowski
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Explore the profile of D Modrowski including associated specialties, affiliations and a list of published articles.
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31
Citations
525
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Recent Articles
1.
Fromigue O, Hay E, Modrowski D, Bouvet S, Jacquel A, Auberger P, et al.
Cell Death Differ
. 2006 Feb;
13(11):1845-56.
PMID: 16470222
Osteosarcoma is the most common primary bone tumour in young adults. Despite improved prognosis, resistance to chemotherapy remains responsible for failure of osteosarcoma treatment. The identification of signals that promote...
2.
Ahdjoudj S, Kaabeche K, Holy X, Fromigue O, Modrowski D, Zerath E, et al.
Exp Cell Res
. 2004 Dec;
303(1):138-47.
PMID: 15572034
The molecular mechanisms regulating the adipogenic differentiation of bone marrow stromal cells in vivo remain largely unknown. In this study, we investigated the regulatory effects of transforming growth factor beta-2...
3.
Delannoy P, Lemonnier J, Hay E, Modrowski D, Marie P
Exp Cell Res
. 2001 Aug;
269(1):154-61.
PMID: 11525648
Cell-cell adhesion mediated by cadherins is believed to play an essential role in the control of cell differentiation and tissue formation. Our recent studies indicate that N-cadherin is involved in...
4.
Lemonnier J, Hay E, Delannoy P, Lomri A, Modrowski D, Caverzasio J, et al.
J Bone Miner Res
. 2001 May;
16(5):832-45.
PMID: 11341328
Apert (Ap) syndrome is characterized by premature cranial suture ossification caused by fibroblast growth factor receptor 2 (FGFR-2) mutations. We studied the role of cadherins and signaling events in the...
5.
Increased osteoblast apoptosis in apert craniosynostosis: role of protein kinase C and interleukin-1
Lemonnier J, Hay E, Delannoy P, Fromigue O, Lomri A, Modrowski D, et al.
Am J Pathol
. 2001 May;
158(5):1833-42.
PMID: 11337381
Apert syndrome is an autosomal dominant disorder characterized by premature cranial ossification resulting from fibroblast growth factor receptor-2 (FGFR-2)-activating mutations. We have studied the effects of the prominent S252W FGFR-2...
6.
Lemonnier J, Delannoy P, Hott M, Lomri A, Modrowski D, Marie P
Exp Cell Res
. 2000 Mar;
256(1):158-67.
PMID: 10739663
We recently showed that the Apert Ser252Trp fibroblast growth factor receptor-2 (FGFR-2) mutation causes premature osteoblast differentiation and increased subperiosteal calvaria bone matrix formation. To gain further insight into the...
7.
Modrowski D, Basle M, Lomri A, Marie P
J Biol Chem
. 2000 Mar;
275(13):9178-85.
PMID: 10734053
We previously showed that granulocyte-macrophage colony-stimulating factor (GM-CSF) binds to heparan sulfate proteoglycans expressed at the surface of osteoblastic cells and that the mitogenic activity of this cytokine is dependent...
8.
Hay E, Lemonnier J, Modrowski D, Lomri A, Lasmoles F, Marie P
J Cell Physiol
. 2000 Mar;
183(1):117-28.
PMID: 10699973
Bone morphogenetic protein-2 (BMP-2) stimulates the differentiation of osteoblastic cells. However, the mechanisms involved in this effect are not well characterized. In this study, we determined the role of the...
9.
Molteni A, Modrowski D, Hott M, Marie P
Bone
. 1999 Apr;
24(4):337-47.
PMID: 10221546
Fibroblast growth factors (FGFs) play important roles in the control of skeletal cell growth and differentiation. To identify the mechanisms of regulation of FGF actions during chondrogenesis and osteogenesis, we...
10.
Molteni A, Modrowski D, Hott M, Marie P
Cell Tissue Res
. 1999 Feb;
295(3):523-36.
PMID: 10022972
Matrix and cell surface proteoglycans (PGs) may play important roles in the control of cellular actions of heparan-binding growth factors such as fibroblast growth factor (FGF) during chondrogenesis and osteogenesis....