Chuansheng Xu
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Explore the profile of Chuansheng Xu including associated specialties, affiliations and a list of published articles.
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10
Citations
163
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Recent Articles
1.
Lin S, Cui Z, Luo Q, Li C, Zhang Y, Yang F, et al.
Adv Sci (Weinh)
. 2025 Jan;
12(9):e2412226.
PMID: 39804981
The healing of bacterial biofilm-infected wounds is a complex process, and the construction of emerging therapeutic modalities that regulate the microenvironment to magnify therapeutic effects and reduce biotoxicity is still...
2.
Zhang Y, Steinmetz-Spah J, Idborg H, Zhu L, Li H, Rao H, et al.
Br J Pharmacol
. 2023 Feb;
180(15):1981-1998.
PMID: 36788645
Background And Purpose: Heart failure with reduced ejection fraction (HFrEF) is a major consequence of myocardial infarction (MI). The microsomal prostaglandin E synthase-1 (mPGES-1)/PGE pathway has been shown to constrain...
3.
Pei J, Cai L, Wang F, Xu C, Pei S, Guo H, et al.
Circ Res
. 2022 Aug;
131(5):388-403.
PMID: 35920162
Rationale: Myocardial infarction (MI) is one of the most dangerous adverse cardiovascular events. Our previous study found that lysophosphatidic acid (LPA) is increased in human peripheral blood after MI, and...
4.
Wan Q, Xu C, Zhu L, Zhang Y, Peng Z, Chen H, et al.
Circ Res
. 2022 Jul;
131(5):442-455.
PMID: 35899614
Background: Timely and complete restoration of blood flow is the most effective intervention for patients with acute myocardial infarction. However, the efficacy is limited by myocardial ischemia-reperfusion (MI/R) injury. PDE4...
5.
Pei S, Xu C, Pei J, Bai R, Peng R, Li T, et al.
Front Immunol
. 2022 Apr;
13:844781.
PMID: 35464399
Sepsis consists of life-threatening organ dysfunction resulting from a dysregulated response to infection. Recent studies have found that excessive neutrophil extracellular traps (NETs) contribute to the pathogenesis of sepsis, thereby...
6.
Chen G, Xu C, Gillette T, Huang T, Huang P, Li Q, et al.
Theranostics
. 2020 Oct;
10(25):11754-11774.
PMID: 33052245
The crosstalk between cardiac microvascular endothelial cells (CMECs) and cardiomyocytes (CMs) has emerged as a key component in the development of, and protection against, cardiac diseases. For example, activation of...
7.
Hao H, Ma X, Chen H, Zhu L, Xu Z, Li Q, et al.
J Cell Mol Med
. 2020 Aug;
24(17):9638-9645.
PMID: 32810369
Neointimal formation and atherogenesis are major vascular complications following percutaneous coronary intervention, and there is lack of pharmacological therapy. This study was aimed to examine the effect of forskolin (FSK),...
8.
Zhu L, Xu C, Huo X, Hao H, Wan Q, Chen H, et al.
Nat Commun
. 2019 Apr;
10(1):1888.
PMID: 31015404
The use of nonsteroidal anti-inflammatory drugs that inhibit cyclooxygenase (COX)-1 and COX-2, increases heart failure risk. It is unknown whether microsomal (m) prostaglandin (PG) E synthase (S)-1, a target downstream...
9.
Chen G, Yang Y, Xu C, Gao S
J Vis Exp
. 2018 Jul;
(137).
PMID: 30059023
Timely and efficient reperfusion of the occluded coronary artery is the best strategy for decreasing myocardial infarct size in patients with a ST-segment elevated myocardial infarction. However, reperfusion per se...
10.
Hao H, Hu S, Wan Q, Xu C, Chen H, Zhu L, et al.
Arterioscler Thromb Vasc Biol
. 2018 Mar;
38(5):1115-1124.
PMID: 29599139
Objective: Deletion of mPGES-1 (microsomal prostaglandin E synthase-1)-an anti-inflammatory target alternative to COX (cyclooxygenase)-2-attenuates injury-induced neointima formation in mice. This is attributable to the augmented levels of PGI (prostacyclin)-a known...