Christina Arieta Kuksin
Overview
Explore the profile of Christina Arieta Kuksin including associated specialties, affiliations and a list of published articles.
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Articles
6
Citations
111
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Recent Articles
1.
Sgolastra F, Kuksin C, Gonzalez-Perez G, Minter L, Tew G
ACS Appl Bio Mater
. 2022 Jan;
1(2):444-451.
PMID: 35016365
Genetic manipulation has increased our understanding of gene function and led to the discovery of new therapeutic targets. Cre/LoxP DNA recombination is widely used for genetic studies in mammalian cells....
2.
Kuksin D, Kuksin C, Qiu J, Chan L
Anal Biochem
. 2016 Apr;
503:1-7.
PMID: 27033005
Traditionally, many cell-based assays that analyze cell populations and functionalities have been performed using flow cytometry. However, flow cytometers remain relatively expensive and require highly trained operators for routine maintenance...
3.
Kuksin C, Minter L
Front Oncol
. 2015 Mar;
5:51.
PMID: 25759795
An association between certain autoimmune conditions and increased risk of developing lymphoma is well documented. Recent evidence points to NOTCH signaling as a strong driver of autoimmunity. Furthermore, a role...
4.
Kuksin C, Gonzalez-Perez G, Minter L
Blood
. 2015 Feb;
125(13):2087-94.
PMID: 25647836
Aplastic anemia (AA) is a disease characterized by T-cell-mediated destruction of bone marrow (BM) hematopoietic stem and progenitor cells. Physiologically, T cells migrate to the BM in response to chemokines,...
5.
Shin H, Tilahun M, Cho O, Chandiran K, Kuksin C, Keerthivasan S, et al.
Front Immunol
. 2014 Jun;
5:249.
PMID: 24904593
T cell stimulation requires the input and integration of external signals. Signaling through the T cell receptor (TCR) is known to induce formation of the membrane-tethered CBM complex, comprising CARMA1,...
6.
Roderick J, Gonzalez-Perez G, Kuksin C, Dongre A, Roberts E, Srinivasan J, et al.
J Exp Med
. 2013 Jun;
210(7):1311-29.
PMID: 23733784
Severe aplastic anemia (AA) is a bone marrow (BM) failure (BMF) disease frequently caused by aberrant immune destruction of blood progenitors. Although a Th1-mediated pathology is well described for AA,...