Celestino Velasquez
Overview
Explore the profile of Celestino Velasquez including associated specialties, affiliations and a list of published articles.
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Articles
5
Citations
139
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0
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Recent Articles
1.
Sun R, Cheng E, Velasquez C, Chang Y, Moore P
J Biol Chem
. 2020 Apr;
295(14):4760.
PMID: 32245903
No abstract available.
2.
Sun R, Cheng E, Velasquez C, Chang Y, Moore P
J Biol Chem
. 2019 Jun;
294(31):11840-11852.
PMID: 31201269
Eukaryotic translation initiation factor 4E (eIF4E)-binding protein 1 (4E-BP1) inhibits cap-dependent translation in eukaryotes by competing with eIF4G for an interaction with eIF4E. Phosphorylation at Ser-83 of 4E-BP1 occurs during...
3.
Velasquez C, Amako Y, Harold A, Toptan T, Chang Y, Shuda M
Front Microbiol
. 2018 Apr;
9:713.
PMID: 29696010
Merkel cell polyomavirus (MCV) plays a causal role in ∼80% of Merkel cell carcinomas (MCC). MCV is clonally integrated into the MCC tumor genome, which results in persistent expression of...
4.
Velasquez C, Cheng E, Shuda M, Lee-Oesterreich P, Pogge von Strandmann L, Gritsenko M, et al.
Proc Natl Acad Sci U S A
. 2016 Jul;
113(30):8466-71.
PMID: 27402756
Mammalian target of rapamycin (mTOR)-directed eukaryotic translation initiation factor 4E-binding protein 1 (4E-BP1) phosphorylation promotes cap-dependent translation and tumorigenesis. During mitosis, cyclin-dependent kinase 1 (CDK1) substitutes for mTOR and fully...
5.
Shuda M, Velasquez C, Cheng E, Cordek D, Kwun H, Chang Y, et al.
Proc Natl Acad Sci U S A
. 2015 Apr;
112(19):5875-82.
PMID: 25883264
Mitosis is commonly thought to be associated with reduced cap-dependent protein translation. Here we show an alternative control mechanism for maintaining cap-dependent translation during mitosis revealed by a viral oncoprotein,...