Camilla Loov
Overview
Explore the profile of Camilla Loov including associated specialties, affiliations and a list of published articles.
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11
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579
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Recent Articles
1.
Hanlon K, Kleinstiver B, Garcia S, Zaborowski M, Volak A, Spirig S, et al.
Nat Commun
. 2019 Oct;
10(1):4439.
PMID: 31570731
Adeno-associated virus (AAV) vectors have shown promising results in preclinical models, but the genomic consequences of transduction with AAV vectors encoding CRISPR-Cas nucleases is still being examined. In this study,...
2.
Gustafsson G, Loov C, Persson E, Lazaro D, Takeda S, Bergstrom J, et al.
Cell Mol Neurobiol
. 2018 Oct;
38(8):1539-1550.
PMID: 30288631
In Parkinson's disease and other Lewy body disorders, the propagation of pathology has been accredited to the spreading of extracellular α-synuclein (α-syn). Although the pathogenic mechanisms are not fully understood,...
3.
Gyorgy B, Loov C, Zaborowski M, Takeda S, Kleinstiver B, Commins C, et al.
Mol Ther Nucleic Acids
. 2018 Jun;
11:429-440.
PMID: 29858078
The APPswe (Swedish) mutation in the amyloid precursor protein (APP) gene causes dominantly inherited Alzheimer's disease (AD) as a result of increased β-secretase cleavage of the amyloid-β (Aβ) precursor protein....
4.
Loov C, Erlandsson A
Methods Mol Biol
. 2017 May;
1594:165-177.
PMID: 28456982
Following cellular engulfment, nanoparticles end up in the lysosomes, making them an ideal tool for modifying the lysosomal environment. Here, we describe how acidic nanoparticles can be used to lower...
5.
Loov C, Scherzer C, Hyman B, Breakefield X, Ingelsson M
Cell Mol Neurobiol
. 2016 Mar;
36(3):437-48.
PMID: 26993503
Fibrillar inclusions of intraneuronal α-synuclein can be detected in certain brain areas from patients with Parkinson's disease (PD) and other disorders with Lewy body pathology. These insoluble protein aggregates do...
6.
Loov C, Mitchell C, Simonsson M, Erlandsson A
Glia
. 2015 Jun;
63(11):1997-2009.
PMID: 26095880
Inefficient lysosomal degradation is central in the development of various brain disorders, but the underlying mechanisms and the involvement of different cell types remains elusive. We have previously shown that...
7.
Loov C, Nadadhur A, Hillered L, Clausen F, Erlandsson A
J Neurotrauma
. 2014 Aug;
32(4):244-51.
PMID: 25087457
Traumatic brain injury (TBI) is a heterogeneous disease, and the discovery of diagnostic and prognostic TBI biomarkers is highly desirable in order to individualize patient care. We have previously published...
8.
Loov C, Shevchenko G, Geeyarpuram Nadadhur A, Clausen F, Hillered L, Wetterhall M, et al.
PLoS One
. 2013 Feb;
8(2):e55983.
PMID: 23409102
The complicated secondary molecular and cellular mechanisms following traumatic brain injury (TBI) are still not fully understood. In the present study, we have used mass spectrometry to identify injury specific...
9.
Loov C, Hillered L, Ebendal T, Erlandsson A
PLoS One
. 2012 Mar;
7(3):e33090.
PMID: 22461890
Clearing of dead cells is a fundamental process to limit tissue damage following brain injury. Engulfment has classically been believed to be performed by professional phagocytes, but recent data show...
10.
Loov C, Fernqvist M, Walmsley A, Marklund N, Erlandsson A
PLoS One
. 2012 Jan;
7(1):e29771.
PMID: 22235341
Identifying external factors that can be used to control neural stem cells division and their differentiation to neurons, astrocytes and oligodendrocytes is of high scientific and clinical interest. Here we...