C D Mills
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Explore the profile of C D Mills including associated specialties, affiliations and a list of published articles.
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46
Citations
1800
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Recent Articles
1.
Mills C, Nguyen T, Tanga F, Zhong C, Gauvin D, Mikusa J, et al.
Eur J Pain
. 2012 Aug;
17(4):469-79.
PMID: 22915527
Background: Injection of nerve growth factor (NGF) produces mechanical and thermal hypersensitivity in rodents and humans. Treatment with sequestering antibodies demonstrates the importance of NGF in various pain states, with...
2.
Zhu C, Mills C, Hsieh G, Zhong C, Mikusa J, LEWIS L, et al.
Eur J Pain
. 2012 Feb;
16(6):816-26.
PMID: 22337256
Background: Most animal models currently used to evaluate antinociceptive efficacy of analgesics rely on the assessment of evoked pain behaviours as primary endpoints. Methods: Here, we have developed and characterized...
3.
Mills C
Crit Rev Immunol
. 2002 Apr;
21(5):399-425.
PMID: 11942557
Macrophages can metabolize arginine to nitric oxide in quantities that inhibit pathogens or nearby host cells. They can instead metabolize arginine to ornithine (a precursor of polyamines and collagen) in...
4.
Mills C, Xu G, McAdoo D, Hulsebosch C
J Neurochem
. 2001 Nov;
79(4):835-48.
PMID: 11723176
Spinal cord injury (SCI) leads to an increase in extracellular excitatory amino acid (EAA) concentrations resulting in glutamate receptor-mediated excitotoxic events. The glutamate receptors include ionotropic (iGluRs) and metabotropic (mGluR)...
5.
Mills C, Grady J, Hulsebosch C
J Neurotrauma
. 2001 Nov;
18(10):1091-105.
PMID: 11686495
Spinal cord injury (SCI) produces abnormal pain syndromes in patients that lead to changes in evoked and spontaneous behaviors. To test if a spontaneous component of pain-like behavior could be...
6.
Mills C, Hains B, Johnson K, Hulsebosch C
J Neurotrauma
. 2001 Aug;
18(8):743-56.
PMID: 11526981
Spinal cord injury (SCI) results in loss of function below the level of injury and the development of chronic central pain (CCP) syndromes. Since different strains may develop and express...
7.
Mills C, Fullwood S, Hulsebosch C
Exp Neurol
. 2001 Jul;
170(2):244-57.
PMID: 11476590
Spinal cord injury (SCI) initiates biochemical events that lead to an increase in extracellular excitatory amino acid concentrations, resulting in glutamate receptor-mediated excitotoxic events. These receptors include the three groups...
8.
Mills C, Xu G, Johnson K, McAdoo D, Hulsebosch C
Neuroreport
. 2000 Oct;
11(14):3067-70.
PMID: 11043525
Spinal cord injury (SCI) leads to an increase in extracellular excitatory amino acid (EAA) concentrations, resulting in glutamate receptor-mediated excitotoxicity and central sensitization. To test contributions of group I metabotropic...
9.
Mills C, Kincaid K, Alt J, Heilman M, Hill A
J Immunol
. 2000 Jun;
164(12):6166-73.
PMID: 10843666
Evidence is provided that macrophages can make M-1 or M-2 responses. The concept of M-1/M-2 fomented from observations that macrophages from prototypical Th1 strains (C57BL/6, B10D2) are more easily activated...
10.
Heilman M, Saxena M, McDermott P, Mills C
J Immunol
. 1999 Nov;
163(11):5877-82.
PMID: 10570272
Evidence is provided that inhibition of macrophage NO production can augment in vivo CTL responses. Specifically, administration of NG-monomethyl-l -arginine (NGMMA) via osmotic pumps increases the tumor-specific CTL response against...