Bryan Bollman
Overview
Explore the profile of Bryan Bollman including associated specialties, affiliations and a list of published articles.
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13
Citations
956
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0
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Recent Articles
1.
Ghezzi L, Tosti V, Shi L, Cantoni C, Mikesell R, Lancia S, et al.
J Neurol Neurosurg Psychiatry
. 2024 Aug;
96(2):158-169.
PMID: 39137977
Background: Calorie restriction (CR) ameliorates preclinical models of multiple sclerosis (MS) via multiple mechanisms. These include decreased leptin, a proinflammatory adipokine, but mechanistic studies in humans are lacking. Tests of...
2.
Holloman J, Dimas S, Archambault A, Filipello F, Du L, Feng J, et al.
Int J Mol Sci
. 2023 Dec;
24(23).
PMID: 38069420
Microglia are found pathologically at all stages of multiple sclerosis (MS) lesion development and are hypothesized to contribute to both inflammatory injury and neuroprotection in the MS brain. Transient receptor...
3.
Filipello F, You S, Mirfakhar F, Mahali S, Bollman B, Acquarone M, et al.
Acta Neuropathol
. 2023 Apr;
145(6):749-772.
PMID: 37115208
TREM2 is an innate immune receptor expressed by microglia in the adult brain. Genetic variation in the TREM2 gene has been implicated in risk for Alzheimer's disease and frontotemporal dementia,...
4.
Ghezzi L, Bollman B, De Feo L, Piccio L, Trapp B, Schmidt R, et al.
Lab Invest
. 2023 Mar;
103(6):100128.
PMID: 36889543
Multiple sclerosis (MS) is a central nervous system (CNS) demyelinating disease. Failure to remyelinate successfully is common in MS lesions, often with consequent neuronal/axonal damage. CNS myelin is normally produced...
5.
Esposito F, Osiceanu A, Sorosina M, Ottoboni L, Bollman B, Santoro S, et al.
Genes (Basel)
. 2022 Dec;
13(12).
PMID: 36553660
While the role of common genetic variants in multiple sclerosis (MS) has been elucidated in large genome-wide association studies, the contribution of rare variants to the disease remains unclear. Herein,...
6.
Manivasagam S, Williams J, Vollmer L, Bollman B, Bartleson J, Ai S, et al.
J Immunol
. 2022 Feb;
208(6):1341-1351.
PMID: 35181638
Type III IFNs (IFNLs) are newly discovered cytokines, acting at epithelial and other barriers, that exert immunomodulatory functions in addition to their primary roles in antiviral defense. In this study,...
7.
Cignarella F, Filipello F, Bollman B, Cantoni C, Locca A, Mikesell R, et al.
Acta Neuropathol
. 2020 Aug;
140(4):513-534.
PMID: 32772264
Multiple sclerosis (MS) is an inflammatory, demyelinating, and neurodegenerative disease of the central nervous system (CNS) triggered by autoimmune mechanisms. Microglia are critical for the clearance of myelin debris in...
8.
Ghezzi L, Cantoni C, Cignarella F, Bollman B, Cross A, Salter A, et al.
Mult Scler
. 2019 Jun;
26(10):1172-1186.
PMID: 31237799
Background: Multiple sclerosis (MS) is a central nervous system (CNS) autoimmune demyelinating disease. Its pathogenesis involves humoral and cellular immunity, with production of pro- and anti-inflammatory cytokines by T cells....
9.
Cantoni C, Cignarella F, Ghezzi L, Mikesell B, Bollman B, Berrien-Elliott M, et al.
Acta Neuropathol
. 2016 Oct;
133(1):61-77.
PMID: 27704281
Myeloid-derived cells play important modulatory and effector roles in multiple sclerosis (MS) and its animal model, experimental autoimmune encephalomyelitis (EAE). Myeloid-derived suppressor cells (MDSCs) are immature myeloid cells, composed of...
10.
Vasek M, Garber C, Dorsey D, Durrant D, Bollman B, Soung A, et al.
Nature
. 2016 Jun;
534(7608):538-43.
PMID: 27337340
Over 50% of patients who survive neuroinvasive infection with West Nile virus (WNV) exhibit chronic cognitive sequelae. Although thousands of cases of WNV-mediated memory dysfunction accrue annually, the mechanisms responsible...