Bernd K Fleischmann
Overview
Explore the profile of Bernd K Fleischmann including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
162
Citations
6762
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Bruckner A, Brandtner A, Rieck S, Fleischmann B, Wenzel D
STAR Protoc
. 2025 Feb;
6(1):103620.
PMID: 39921861
Endothelial cells (ECs) play a major role in vascular homeostasis and pathology. They show a pronounced heterogeneity between different organs as well as within the same organ. Here, we present...
2.
Risse I, Filippi K, Wiemann M, Fleischmann B, Hesse M
Stem Cell Res
. 2024 Dec;
82():103641.
PMID: 39729860
BAG3 contributes to the maintenance of proteostasis through chaperone-assisted selective autophagy. This function is impaired by a single amino acid exchange (P209L) in the protein, which causes myofibrillar myopathy-6 (MFM6)....
3.
Filippi K, Wiemann M, Fleischmann B, Hesse M
Stem Cell Res
. 2024 Dec;
82():103627.
PMID: 39662463
BAG3 plays a key role in proteostasis as a central component of the chaperone-assisted selective autophagy (CASA) complex. A point mutation (p.P209L; c.626C>T) in the BAG3 gene causes severe myofibrillar...
4.
Filippi K, Risse I, Judge L, Conklin B, Fleischmann B, Hesse M
Stem Cell Res
. 2024 Dec;
82():103626.
PMID: 39662461
BAG3 is a central component of the chaperone-assisted selective autophagy complex and thus important for proteostasis. This function is affected by a point mutation (p.P209L; c.626C>T) in the BAG3 gene,...
5.
Hu T, Fleischmann B, Malek Mohammadi M
Lab Anim (NY)
. 2024 Oct;
53(11):308-326.
PMID: 39438662
The adult mammalian heart is known to have very limited regenerative capacity, explaining at least in part the frequency of cardiovascular diseases and their impact as the leading cause of...
6.
Insulin-Activated Signaling Pathway and GLUT4 Membrane Translocation in hiPSC-Derived Cardiomyocytes
Querio G, Antoniotti S, Levi R, Fleischmann B, Gallo M, Malan D
Int J Mol Sci
. 2024 Aug;
25(15).
PMID: 39125765
Human induced pluripotent stem cell-derived cardiomyocytes (hiPSC-CM) are a cell model now widely used to investigate pathophysiological features of cardiac tissue. Given the invaluable contribution hiPSC-CM could make for studies...
7.
Seidinger A, Roberts R, Bai Y, Muller M, Pfeil E, Matthey M, et al.
EMBO Mol Med
. 2024 Jul;
16(8):1930-1956.
PMID: 38977926
Pulmonary arterial hypertension (PAH) is a life-threatening disease with limited survival. Herein, we propose the pharmacological inhibition of Gq proteins as a novel concept to counteract pulmonary vasoconstriction and proliferation/migration...
8.
Bruckner A, Brandtner A, Rieck S, Matthey M, Geisen C, Fels B, et al.
Angiogenesis
. 2024 Jul;
27(4):719-738.
PMID: 38965173
Aortic aneurysm is characterized by a pathological dilation at specific predilection sites of the vessel and potentially results in life-threatening vascular rupture. Herein, we established a modified "Häutchen method" for...
9.
Rieck S, Sharma K, Altringer C, Hesse M, Triantafyllou C, Zhang Y, et al.
Cardiovasc Res
. 2024 Jun;
120(12):1472-1484.
PMID: 38916487
Aims: Endothelial cell (EC) dysfunction plays a key role in the initiation and progression of cardiovascular disease. However, studying these disorders in ECs from patients is challenging; hence, the use...
10.
Roshanbinfar K, Schiffer M, Carls E, Angeloni M, Kolesnik-Gray M, Schruefer S, et al.
Adv Mater
. 2024 Apr;
36(28):e2403642.
PMID: 38653478
Myocardial infarction (MI) causes cell death, disrupts electrical activity, triggers arrhythmia, and results in heart failure, whereby 50-60% of MI-associated deaths manifest as sudden cardiac deaths (SCD). The most effective...