Benjamin P Sinder
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Explore the profile of Benjamin P Sinder including associated specialties, affiliations and a list of published articles.
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Articles
16
Citations
459
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0
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Recent Articles
1.
Parathyroid hormone and trabectedin have differing effects on macrophages and stress fracture repair
Zweifler L, Sinder B, Stephan C, Koh A, Do J, Ulrich E, et al.
Bone
. 2023 Nov;
179:116983.
PMID: 38013019
Stress fractures occur as a result of repeated mechanical stress on bone and are commonly found in the load-bearing lower extremities. Macrophages are key players in the immune system and...
2.
Ahn T, Jueckstock M, Mandair G, Henderson J, Sinder B, Kozloff K, et al.
J Struct Biol
. 2022 Jul;
214(3):107878.
PMID: 35781024
Atomic force microscopy-infrared spectroscopy (AFM-IR) and optical photothermal infrared spectroscopy (O-PTIR), which feature spectroscopic imaging spatial resolution down to ∼ 50 nm and ∼ 500 nm, respectively, were employed to...
3.
Wee N, Vrhovac Madunic I, Ivanisevic T, Sinder B, Kalajzic I
J Musculoskelet Neuronal Interact
. 2020 Dec;
20(4):579-590.
PMID: 33265087
Objectives: Neuropeptide Y (NPY) is involved in the coordination of bone mass and adiposity. However, multiple NPY sources exist and their individual contribution to the skeleton and adiposity not known....
4.
Novak S, Roeder E, Sinder B, Adams D, Siebel C, Grcevic D, et al.
J Orthop Res
. 2020 Mar;
38(11):2350-2361.
PMID: 32141629
Fracture healing involves interactions of different cell types, driven by various growth factors, and signaling cascades. Periosteal mesenchymal progenitor cells give rise to the majority of osteoblasts and chondrocytes in...
5.
Sinder B, Novak S, Wee N, Basile M, Maye P, Matthews B, et al.
Stem Cells
. 2019 Dec;
38(4):530-541.
PMID: 31859429
Osteogenesis imperfecta (OI) is a genetic disorder most commonly caused by mutations associated with type I collagen, resulting in a defective collagen bone matrix. Current treatments for OI focus on...
6.
Wee N, Sinder B, Novak S, Wang X, Stoddard C, Matthews B, et al.
Neuropeptides
. 2018 Dec;
73:78-88.
PMID: 30522780
Neuropeptide Y (NPY) is involved in multiple processes such as behavior, energy and bone metabolism. Previous studies have relied on global NPY depletion to examine its effects on bone. However,...
7.
Michalski M, Seydel A, Siismets E, Zweifler L, Koh A, Sinder B, et al.
FASEB J
. 2018 Feb;
32(7):3730-3741.
PMID: 29475373
A prolonged increase in proinflammatory cytokines is associated with osteoporotic and autoimmune bone loss and, conversely, anti-inflammatory pathways are associated with protection against bone loss. Milk fat globule-epidermal growth factor...
8.
Sinder B, Zweifler L, Koh A, Michalski M, Hofbauer L, Aguirre J, et al.
J Bone Miner Res
. 2017 Jun;
32(10):2116-2127.
PMID: 28600866
Macrophages have established roles supporting bone formation. Despite their professional phagocytic nature, the role of macrophage phagocytosis in bone homeostasis is not well understood. Interestingly, apoptosis is a pivotal feature...
9.
Perosky J, Khoury B, Jenks T, Ward F, Cortright K, Meyer B, et al.
Bone
. 2016 Sep;
93:79-85.
PMID: 27641475
Sclerostin antibody has demonstrated a bone-forming effect in pre-clinical models of osteogenesis imperfecta, where mutations in collagen or collagen-associated proteins often result in high bone fragility in pediatric patients. Cessation...
10.
Sinder B, Lloyd W, Salemi J, Marini J, Caird M, Morris M, et al.
Bone
. 2016 Jan;
84:222-229.
PMID: 26769006
Bone composition and biomechanics at the tissue-level are important contributors to whole bone strength. Sclerostin antibody (Scl-Ab) is a candidate anabolic therapy for the treatment of osteoporosis that increases bone...