B Annabi
Overview
Explore the profile of B Annabi including associated specialties, affiliations and a list of published articles.
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Articles
16
Citations
217
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0
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Recent Articles
1.
Unger B, Gillman L, Hickey K, Pace D, Lam-Tin-Cheung K, Catton J, et al.
Can J Surg
. 2022 Apr;
64(6 Suppl 2):S80-S159.
PMID: 35483046
No abstract available.
2.
Gingras D, Langlois S, Lachambre M, Annabi B, Beliveau R
FEBS Lett
. 2001 Oct;
507(2):231-6.
PMID: 11684104
The mechanisms underlying membrane-type-1 matrix metalloproteinase (MT1-MMP)-dependent induction of cell migration were investigated. Overexpression of MT1-MMP induced a marked increase in cell migration, this increase being dependent on the presence...
3.
Annabi B, Pilorget A, Gingras D, Beliveau R
Biochem J
. 2001 Oct;
359(Pt 2):325-33.
PMID: 11583578
Most transmembrane proteins are subjected to limited proteolysis by cellular proteases, and stimulation of cleavage of membrane proteins by calmodulin (CaM) inhibitors was recently shown. The present study investigated the...
4.
Annabi B, Lachambre M, Page M, Gingras D, Beliveau R
Biochem J
. 2001 Feb;
353(Pt 3):547-53.
PMID: 11171051
Membrane-type 1 matrix metalloproteinase (MT1-MMP) is a membrane-associated MMP that has been recently reported to have a central role in tumour cell invasion. Here we report that both the native...
5.
Mechin M, Annabi B, Pegorier J, van de Werve G
Metabolism
. 2000 Oct;
49(9):1200-3.
PMID: 11016904
The catalytic subunit (p36) and putative glucose-6-phosphate (G6P) transporter (p46) protein levels of the rat glucose-6-phosphatase (G6Pase) system were studied in relation to G6Pase hydrolytic activity and G6P uptake in...
6.
Gingras D, Page M, Annabi B, Beliveau R
Biochim Biophys Acta
. 2000 Sep;
1497(3):341-50.
PMID: 10996658
Matrix metalloproteinase-2 (MMP-2) has been suggested to play a crucial role in tumor invasion and angiogenesis. In order to understand the mechanisms underlying proMMP-2 activation, we compared the biochemical and...
7.
Lin B, Annabi B, Hiraiwa H, Pan C, Chou J
J Biol Chem
. 1998 Nov;
273(48):31656-60.
PMID: 9822626
Glycogen storage disease type 1 (GSD-1) is a group of genetic disorders caused by a deficiency in the activity of the enzyme glucose-6-phosphatase. (G6Pase). GSD-1a and GSD-1b, the two major...
8.
Pan C, Lei K, Annabi B, Hemrika W, Chou J
J Biol Chem
. 1998 Apr;
273(11):6144-8.
PMID: 9497333
Deficiency of microsomal glucose-6-phosphatase (G6Pase), the key enzyme in glucose homeostasis, causes glycogen storage disease type 1a, an autosomal recessive disorder. Characterization of the transmembrane topology of G6Pase should facilitate...
9.
Annabi B, Hiraiwa H, Mansfield B, Lei K, Ubagai T, Polymeropoulos M, et al.
Am J Hum Genet
. 1998 Apr;
62(2):400-5.
PMID: 9463334
Glycogen-storage disease type 1 (GSD-1), also known as "von Gierke disease," is caused by a deficiency in microsomal glucose-6-phosphatase (G6Pase) activity. There are four distinct subgroups of this autosomal recessive...
10.
Annabi B, van de Werve G
Biochem Biophys Res Commun
. 1997 Jul;
236(3):808-13.
PMID: 9245738
We show that the production of glucose from glucose-6-phosphate hydrolysis outside microsomes is a function of glucose-6-phosphatase independent of its property to form glucose inside microsomes. Indeed, during development (before...