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Ashlee E Tyler

Explore the profile of Ashlee E Tyler including associated specialties, affiliations and a list of published articles. Areas
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Articles 6
Citations 282
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Recent Articles
1.
Diede S, Yao Z, Keyes C, Tyler A, Dey J, Hackett C, et al.
Epigenetics . 2013 Oct; 8(12):1254-60. PMID: 24107773
Genetic and epigenetic alterations are essential for the initiation and progression of human cancer. We previously reported that primary human medulloblastomas showed extensive cancer-specific CpG island DNA hypermethylation in critical...
2.
Parker M, Loretz C, Tyler A, Duddy W, Hall J, Olwin B, et al.
Stem Cells . 2012 Aug; 30(10):2212-20. PMID: 22865615
Transplantation of myogenic stem cells possesses great potential for long-term repair of dystrophic muscle. However, a single donor muscle biopsy is unlikely to provide enough cells to effectively transplant the...
3.
Parker M, Loretz C, Tyler A, Snider L, Storb R, Tapscott S
Skelet Muscle . 2012 Feb; 2(1):4. PMID: 22340947
Background: Transplantation of myogenic stem cells possesses great potential for long-term repair of dystrophic muscle. In murine-to-murine transplantation experiments, CXCR4 expression marks a population of adult murine satellite cells with...
4.
Geng L, Tyler A, Tapscott S
Hybridoma (Larchmt) . 2011 May; 30(2):125-30. PMID: 21529284
Double homeobox 4 (DUX4) is a candidate disease gene for facioscapulohumeral dystrophy (FSHD), one of the most common muscular dystrophies characterized by progressive skeletal muscle degeneration. Despite great strides in...
5.
Snider L, Asawachaicharn A, Tyler A, Geng L, Petek L, Maves L, et al.
Hum Mol Genet . 2009 Apr; 18(13):2414-30. PMID: 19359275
Deletion of a subset of the D4Z4 macrosatellite repeats in the subtelomeric region of chromosome 4q causes facioscapulohumeral muscular dystrophy (FSHD) when occurring on a specific haplotype of 4qter (4qA161)....
6.
Yang Z, MacQuarrie K, Analau E, Tyler A, Dilworth F, Cao Y, et al.
Genes Dev . 2009 Mar; 23(6):694-707. PMID: 19299559
Rhabdomyosarcomas are characterized by expression of myogenic specification genes, such as MyoD and/or Myf5, and some muscle structural genes in a population of cells that continues to replicate. Because MyoD...