Antonello Lorenzini
Overview
Explore the profile of Antonello Lorenzini including associated specialties, affiliations and a list of published articles.
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38
Citations
867
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Recent Articles
31.
Lorenzini A, Stamato T, Sell C
Cell Cycle
. 2011 Nov;
10(22):3853-6.
PMID: 22071624
All life processes are subject to time constraints. At the cellular level, damage repair and cell cycle arrest are interrelated, allowing sufficient time for repair prior to cell cycle progression....
32.
Fink L, Roell M, Caiazza E, Lerner C, Stamato T, Hrelia S, et al.
Aging (Albany NY)
. 2011 Sep;
3(9):836-45.
PMID: 21931182
Faithful repair of damaged DNA is a crucial process in maintaining cell viability and function. A multitude of factors and pathways guides this process and includes repair proteins and cell...
33.
Angeloni C, Motori E, Fabbri D, Malaguti M, Leoncini E, Lorenzini A, et al.
Am J Physiol Heart Circ Physiol
. 2011 Apr;
300(6):H2196-205.
PMID: 21478407
Ischemic preconditioning is a complex cardioprotective phenomenon that involves adaptive changes in cells and molecules and occurs in a biphasic pattern: an early phase after 1-2 h and a late...
34.
Bitto A, Lerner C, Torres C, Roell M, Malaguti M, Perez V, et al.
PLoS One
. 2010 Sep;
5(9):e12592.
PMID: 20830296
A reduction in IGF-I signaling has been found to increase lifespan in multiple organisms despite the fact that IGF-I is a trophic factor for many cell types and has been...
35.
Bitto A, Sell C, Crowe E, Lorenzini A, Malaguti M, Hrelia S, et al.
Exp Cell Res
. 2010 Jul;
316(17):2961-8.
PMID: 20620137
There is an increasing awareness that astrocytes, the most abundant cell type in the central nervous system, are critical mediators of brain homeostasis, playing multifunctional roles including buffering potassium ions,...
36.
Lorenzini A, Johnson F, Oliver A, Tresini M, Smith J, Hdeib M, et al.
Mech Ageing Dev
. 2009 Nov;
130(11-12):784-92.
PMID: 19896964
The identification of the cellular mechanisms responsible for the wide differences in species lifespan remains one of the major unsolved problems of the biology of aging. We measured the capacity...
37.
Tresini M, Lorenzini A, Torres C, Cristofalo V
J Biol Chem
. 2006 Dec;
282(6):4136-51.
PMID: 17145763
Normal somatic cells have a limited replicative lifespan, and serial subcultivation ultimately results in senescence. Senescent cells are irreversibly growth-arrested and show impaired responses to mitogens. Activation of the ERK...
38.
Lorenzini A, Tresini M, Austad S, Cristofalo V
Mech Ageing Dev
. 2005 Jul;
126(10):1130-3.
PMID: 15993927
Although the limited replicative capacity of human fibroblasts in culture is frequently used as a model for aging, a question of major interest is whether the relationship between in vitro...
39.
Cristofalo V, Lorenzini A, Allen R, Torres C, Tresini M
Mech Ageing Dev
. 2004 Nov;
125(10-11):827-48.
PMID: 15541776
Human cells in culture have a limited proliferative capacity. After a period of vigorous proliferation, the rate of cell division declines and a number of changes occur in the cells...
40.
Torres C, Francis M, Lorenzini A, Tresini M, Cristofalo V
Exp Cell Res
. 2003 Oct;
290(2):195-206.
PMID: 14567979
Cellular senescence is characterized by impaired cell proliferation. We have previously shown that, relative to the young counterpart, senescent WI-38 human fibroblasts display a decreased abundance of active phosphorylated ERK...