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Anne Bonnieu

Explore the profile of Anne Bonnieu including associated specialties, affiliations and a list of published articles. Areas
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Articles 32
Citations 696
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Recent Articles
11.
Stantzou A, Ueberschlag-Pitiot V, Thomasson R, Furling D, Bonnieu A, Amthor H, et al.
Muscle Nerve . 2016 Jun; 55(2):254-261. PMID: 27312354
Introduction: The effect of constitutive inactivation of the gene encoding myostatin on the gain in muscle performance during postnatal growth has not been well characterized. Methods: We analyzed 2 murine...
12.
Pagano A, Demangel R, Brioche T, Jublanc E, Bertrand-Gaday C, Candau R, et al.
PLoS One . 2015 Dec; 10(12):e0144230. PMID: 26629696
Sports trauma are able to induce muscle injury with fibrosis and accumulation of intermuscular adipose tissue (IMAT), which affect muscle function. This study was designed to investigate whether hypoactivity would...
13.
Philippe A, Py G, Favier F, Sanchez A, Bonnieu A, Busso T, et al.
Biomed Res Int . 2015 Feb; 2015:914860. PMID: 25695093
The aim of the present study was to test whether systems models of training effects on performance in athletes can be used to explore the responses to resistance training in...
14.
Gallot Y, Durieux A, Castells J, Desgeorges M, Vernus B, Plantureux L, et al.
Cancer Res . 2014 Oct; 74(24):7344-56. PMID: 25336187
Cachexia is a muscle-wasting syndrome that contributes significantly to morbidity and mortality of many patients with advanced cancers. However, little is understood about how the severe loss of skeletal muscle...
15.
Britto F, Begue G, Rossano B, Docquier A, Vernus B, Sar C, et al.
Am J Physiol Endocrinol Metab . 2014 Oct; 307(11):E983-93. PMID: 25315696
REDD1 (regulated in development and DNA damage response 1) has been proposed to inhibit the mechanistic target of rapamycin complex 1 (mTORC1) during in vitro hypoxia. REDD1 expression is low...
16.
Brun C, Perie L, Baraige F, Vernus B, Bonnieu A, Blanquet V
Cell Physiol Biochem . 2014 Oct; 34(4):1241-59. PMID: 25277978
Background/aims: Overexpression of Gasp-1, an inhibitor of myostatin, leads to a hypermuscular phenotype due to hypertrophy rather than hyperplasia in mice. However to date, the cellular and molecular mechanisms underlying...
17.
Pauly M, Chabi B, Favier F, Vanterpool F, Matecki S, Fouret G, et al.
J Gerontol A Biol Sci Med Sci . 2014 Sep; 70(9):1077-87. PMID: 25227129
Myostatin (mstn) blockade, resulting in muscle hypertrophy, is a promising therapy to counteract age-related muscle loss. However, oxidative and mitochondrial deficit observed in young mice with myostatin inhibition could be...
18.
Haddad M, Jean E, Turki A, Hugon G, Vernus B, Bonnieu A, et al.
J Cell Sci . 2012 Nov; 125(Pt 24):6147-56. PMID: 23132926
Protection of satellite cells from cytotoxic damages is crucial to ensure efficient adult skeletal muscle regeneration and to improve therapeutic efficacy of cell transplantation in degenerative skeletal muscle diseases. It...
19.
Gay S, Jublanc E, Bonnieu A, Bacou F
Muscle Nerve . 2012 Apr; 45(5):698-704. PMID: 22499097
Introduction: Myostatin (Mstn) is a secreted protein that acts as a negative regulator of skeletal muscle mass. However, a critical evaluation of neuromuscular aspects of hypertrophied muscles induced by Mstn...
20.
Ploquin C, Chabi B, Fouret G, Vernus B, Feillet-Coudray C, Coudray C, et al.
Am J Physiol Endocrinol Metab . 2012 Feb; 302(8):E1000-8. PMID: 22318951
Loss of myostatin (mstn) function leads to a decrease in mitochondrial content, a reduced expression of cytochrome c oxidase, and a lower citrate synthase activity in skeletal muscle. These data...