Andrew Cant
Overview
Explore the profile of Andrew Cant including associated specialties, affiliations and a list of published articles.
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51
Citations
2356
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Recent Articles
11.
Lum S, Flood T, Hambleton S, McNaughton P, Watson H, Abinun M, et al.
Blood
. 2019 Apr;
133(23):2546-2549.
PMID: 30952673
No abstract available.
12.
Bucciol G, Nicholas S, Calvo P, Cant A, Edgar J, Espanol T, et al.
J Allergy Clin Immunol
. 2019 Jan;
143(5):1952-1956.e6.
PMID: 30682461
No abstract available.
13.
Shahin T, Aschenbrenner D, Cagdas D, Bal S, Conde C, Garncarz W, et al.
Haematologica
. 2018 Oct;
104(3):609-621.
PMID: 30309848
Hyper-IgE syndromes comprise a group of inborn errors of immunity. STAT3-deficient hyper-IgE syndrome is characterized by elevated serum IgE levels, recurrent infections and eczema, and characteristic skeletal anomalies. A loss-of-function...
14.
Schwab C, Gabrysch A, Olbrich P, Patino V, Warnatz K, Wolff D, et al.
J Allergy Clin Immunol
. 2018 May;
142(6):1932-1946.
PMID: 29729943
Background: Cytotoxic T-lymphocyte antigen 4 (CTLA-4) is a negative immune regulator. Heterozygous CTLA4 germline mutations can cause a complex immune dysregulation syndrome in human subjects. Objective: We sought to characterize...
15.
Maccari M, Abolhassani H, Aghamohammadi A, Aiuti A, Aleinikova O, Bangs C, et al.
Front Immunol
. 2018 Mar;
9:543.
PMID: 29599784
Activated phosphoinositide 3-kinase (PI3K) δ Syndrome (APDS), caused by autosomal dominant mutations in (APDS1) or (APDS2), is a heterogeneous primary immunodeficiency. While initial cohort-descriptions summarized the spectrum of clinical and...
16.
Altmann T, Slack J, Slatter M, OBrien C, Cant A, Thomas M, et al.
Bone Marrow Transplant
. 2018 Jan;
53(4):515-518.
PMID: 29335628
No abstract available.
17.
Bigley V, Maisuria S, Cytlak U, Jardine L, Care M, Green K, et al.
J Allergy Clin Immunol
. 2017 Nov;
141(6):2234-2248.
PMID: 29128673
Background: The homozygous K108E mutation of interferon regulatory factor 8 (IRF8) is reported to cause dendritic cell (DC) and monocyte deficiency. However, more widespread immune dysfunction is predicted from the...
18.
Schwerd T, Twigg S, Aschenbrenner D, Manrique S, Miller K, Taylor I, et al.
J Exp Med
. 2017 Jul;
214(9):2547-2562.
PMID: 28747427
Multiple cytokines, including interleukin 6 (IL-6), IL-11, IL-27, oncostatin M (OSM), and leukemia inhibitory factor (LIF), signal via the common GP130 cytokine receptor subunit. In this study, we describe a...
19.
Mahlaoui N, Warnatz K, Jones A, Workman S, Cant A
J Clin Immunol
. 2017 May;
37(5):452-460.
PMID: 28523402
Primary immunodeficiencies (PIDs) are a widely heterogeneous group of inherited defects of the immune system consisting of many clinical phenotypes with at least 300 underlying genetic deficits currently known. Patients...
20.
de la Morena M, Leonard D, Torgerson T, Cabral-Marques O, Slatter M, Aghamohammadi A, et al.
J Allergy Clin Immunol
. 2016 Oct;
139(4):1282-1292.
PMID: 27697500
Background: X-linked hyper-IgM syndrome (XHIGM) is a primary immunodeficiency with high morbidity and mortality compared with those seen in healthy subjects. Hematopoietic cell transplantation (HCT) has been considered a curative...