Ana Cenarro
Overview
Explore the profile of Ana Cenarro including associated specialties, affiliations and a list of published articles.
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Articles
104
Citations
994
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Recent Articles
1.
Rodriguez-Hernandez Z, Bel-Aguilar J, Moreno-Franco B, Grau-Perez M, Redon J, Gomez-Ariza J, et al.
Nutr Diabetes
. 2025 Feb;
15(1):5.
PMID: 39948355
Objective: To assess whether the role of selenium on pre-diabetes is differential by age, given comorbidities and decreased β-cell function in older adults. Research Design And Methods: We evaluated the...
2.
La Chica Lhoest M, Martinez A, Garcia E, Dandurand J, Polishchuk A, Benitez-Amaro A, et al.
J Lipid Res
. 2024 Nov;
66(1):100703.
PMID: 39557294
Patients with familial hypercholesterolemia (FH) exhibit a significant residual cardiovascular risk. A new cardiovascular risk factor is the susceptibility of individual LDL particles to aggregation. This study examined LDL aggregation...
3.
Bea A, Gonzalez-Guerrero A, Cenarro A, Lamiquiz-Moneo I, Climent E, Jarauta E, et al.
Atherosclerosis
. 2024 Oct;
400:118617.
PMID: 39368903
Background And Aims: Recent reports have shown that subjects with high high-density lipoprotein cholesterol (HDLc) levels are paradoxically at increased risk for all-cause and cardiovascular mortality. The aim was to...
4.
Saenz-Pipaon G, Cenarro A, Zazpe J, Goni-Oloriz M, Martinez-Aguilar E, Machado F, et al.
Clin Investig Arterioscler
. 2024 Sep;
36(6):343-355.
PMID: 39271430
Introduction: Despite the key role of the endothelium in atherosclerosis, there are no direct techniques for its analysis. The study of extracellular vesicles of endothelial origin (EEVs), might lead to...
5.
Paramo J, Cenarro A, Civeira F, Roncal C
Clin Investig Arterioscler
. 2024 May;
37(2):100718.
PMID: 38714381
Atherosclerosis is the main pathogenic substrate for cardiovascular diseases (CVDs). Initially categorized as a passive cholesterol storage disease, nowadays, it is considered an active process, identifying inflammation among the key...
6.
Civeira F, Martin C, Cenarro A
Curr Opin Lipidol
. 2024 Apr;
35(4):195-199.
PMID: 38640077
Purpose Of Review: Autosomal dominant hypercholesterolemia is a common cause of cardiovascular disease. In addition to the classic genes that cause hypercholesterolemia, LDLR, APOB and PCSK9 , a new locus...
7.
Larrea-Sebal A, Sasiain I, Jebari-Benslaiman S, Galicia-Garcia U, Uribe K, Benito-Vicente A, et al.
Adv Sci (Weinh)
. 2024 Jan;
11(13):e2305177.
PMID: 38258479
Familial hypercholesterolemia (FH) is an inherited metabolic disease affecting cholesterol metabolism, with 90% of cases caused by mutations in the LDL receptor gene (LDLR), primarily missense mutations. This study aims...
8.
Corredoira P, Marco-Benedi V, Cenarro A, Peribanez S, Olmos S, Civeira F
Rev Esp Cardiol (Engl Ed)
. 2024 Jan;
77(6):462-470.
PMID: 38185215
Introduction And Objectives: Tendon xanthomas (TX) are lipid deposits highly specific to familial hypercholesterolemia (FH). However, there is significant variability in their presentation among FH patients, primarily due to largely...
9.
Marco-Benedi V, Cenarro A, Laclaustra M, Calmarza P, Bea A, Vila A, et al.
Clin Investig Arterioscler
. 2023 Dec;
36(2):71-77.
PMID: 38161102
Background: Recently, an inverse relationship between the blood concentration of lipoprotein(a) (Lp(a)) and triglycerides (TG) has been demonstrated. The larger the VLDL particle size, the greater the presence of VLDL...
10.
Marco-Benedi V, Cenarro A, Vila A, Real J, Tamarit J, Walther L, et al.
J Clin Lipidol
. 2023 Oct;
17(6):717-731.
PMID: 37813710
Background: Clinically diagnosed familial hypercholesterolemia (FH) may require a genetic test (GT) to confirm diagnosis. GT availability/accessibility is resource-dependent and usually restricted to specialized clinics. While GT has a diagnostic...