Amy L Silvers
Overview
Explore the profile of Amy L Silvers including associated specialties, affiliations and a list of published articles.
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9
Citations
315
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Recent Articles
1.
Lin L, Bass A, Lockwood W, Wang Z, Silvers A, Thomas D, et al.
Proc Natl Acad Sci U S A
. 2012 Mar;
109(11):4251-6.
PMID: 22375031
Gene amplification is a tumor-specific event during malignant transformation. Recent studies have proposed a lineage-dependency (addiction) model of human cancer whereby amplification of certain lineage transcription factors predisposes a survival...
2.
Hartojo W, Silvers A, Thomas D, Seder C, Lin L, Rao H, et al.
Transl Oncol
. 2010 Apr;
3(2):99-108.
PMID: 20360934
The transcription factor, nuclear factor kappaB (NF-kappaB), plays a central role as a key mediator of cell survival and proliferation, and its activation may confer increased tumor chemoresistance. Curcumin, an...
3.
Silvers A, Lin L, Bass A, Chen G, Wang Z, Thomas D, et al.
Clin Cancer Res
. 2010 Mar;
16(7):2009-21.
PMID: 20332323
Purpose: The chemopreventive effects of selenium have been extensively examined, but its role in cancer development or as a chemotherapeutic agent has only recently been explored. Because selenium-binding protein 1...
4.
Seder C, Hartojo W, Lin L, Silvers A, Wang Z, Thomas D, et al.
Neoplasia
. 2009 Mar;
11(4):388-96.
PMID: 19308293
Introduction: The expression, mechanisms of regulation, and functional impact of INHBA (activin A) in lung adenocarcinoma (AD) have not been fully elucidated. Methods: INHBA expression was examined in 96 lung...
5.
Seder C, Hartojo W, Lin L, Silvers A, Wang Z, Thomas D, et al.
J Thorac Oncol
. 2009 Feb;
4(4):455-62.
PMID: 19240652
Introduction: The expression, mechanisms of regulation, and functional impact of activin (INHBA) in esophageal adenocarcinoma (EAC) have not been fully defined. Methods: INHBA expression was examined in 46 esophageal samples...
6.
Silvers A, Finch J, Timothy Bowden G
Photochem Photobiol
. 2006 Apr;
82(2):423-31.
PMID: 16613495
Inhibition of c-Jun N-terminal kinase (JNK) with the pharmacologic inhibitor SP600125 in UVA-irradiated HaCaT cells and human primary keratinocytes resulted in dramatic phenotypic changes indicative of cell death. These phenotypic...
7.
Silvers A, Bachelor M, Timothy Bowden G
Neoplasia
. 2003 Sep;
5(4):319-29.
PMID: 14511403
To further delineate ultraviolet A (UVA) signaling pathways in the human keratinocyte cell line HaCaT, we examined the potential role of mitogen-activated protein kinases (MAPKs) in UVA-induced activator protein-1 (AP-1)...
8.
Bachelor M, Silvers A, Bowden G
Oncogene
. 2002 Oct;
21(46):7092-9.
PMID: 12370831
We examined the expression of cycloxygenase-2, the rate-limiting enzyme in the production of prostaglandins, in the UVA-irradiated human keratinocyte cell line, HaCaT. UVA induced a dose-dependent increase in COX-2 at...
9.
Silvers A, Timothy Bowden G
Photochem Photobiol
. 2002 Apr;
75(3):302-10.
PMID: 11950097
To determine whether the transcription factor activator protein-1 (AP-1) could be modulated by ultraviolet A (UVA) exposure, we examined AP-1 DNA-binding activity and transactivation after exposure to UVA in the...