Alisa Forster
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Explore the profile of Alisa Forster including associated specialties, affiliations and a list of published articles.
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Articles
11
Citations
77
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Recent Articles
1.
Junk S, Forster A, Schmidt G, Zimmermann M, Fedders B, Haermeyer B, et al.
Leukemia
. 2024 Feb;
38(4):887-892.
PMID: 38413718
No abstract available.
2.
Weber C, Kronke N, Volk V, Auber B, Forster A, Trost D, et al.
Acta Neuropathol Commun
. 2023 Nov;
11(1):184.
PMID: 37990341
Pathogenic germline variants in the DNA polymerase genes POLE and POLD1 cause polymerase proofreading-associated polyposis, a dominantly inherited disorder with increased risk of colorectal carcinomas and other tumors. POLE/POLD1 variants...
3.
Forster A, Davenport C, Duployez N, Erlacher M, Ferster A, Fitzgibbon J, et al.
Eur J Med Genet
. 2023 Feb;
66(4):104727.
PMID: 36775010
Although hematologic malignancies (HM) are no longer considered exclusively sporadic, additional awareness of familial cases has yet to be created. Individuals carrying a (likely) pathogenic germline variant (e.g., in ETV6,...
4.
Forster A, Decker M, Schlegelberger B, Ripperger T
Cancers (Basel)
. 2022 Jul;
14(14).
PMID: 35884491
Pathogenic loss-of-function germline variants cause autosomal dominantly-inherited familial platelet disorder with predisposition to hematologic malignancies (RUNX1-FPD). RUNX1-FPD is characterized by incomplete penetrance and a broad spectrum of clinical phenotypes, even...
5.
Osmanovic A, Forster A, Widjaja M, Auber B, Das A, Christians A, et al.
J Neurol
. 2022 May;
269(9):4863-4871.
PMID: 35503374
Background: Recent evidence points toward a role of the small ubiquitin-like modifier (SUMO) system, including SUMO4, in protecting from stress insults and neurodegeneration, such as the progressive motor neuron disease...
6.
Basenach E, Forster A, Raab P, Alzein S, Schmidt G, Krauss J, et al.
Clin Neuropathol
. 2022 Apr;
41(4):162-167.
PMID: 35445657
Neurofibromatosis type 2 (NF2) is a tumor predisposition syndrome characterized by the growth of schwannomas, especially bilateral vestibular schwannomas (VS), meningiomas, and ependymomas. The anti-VEGF antibody bevacizumab has shown efficacy...
7.
Decker M, Agarwal A, Benneche A, Churpek J, Duployez N, Duvall A, et al.
Blood Adv
. 2022 Jan;
6(11):3195-3200.
PMID: 35026845
Familial platelet disorder with associated myeloid malignancies (RUNX1-familial platelet disorder [RUNX1-FPD]) is caused by heterozygous pathogenic germline variants of RUNX1. In the present study, we evaluate the applicability of transactivation...
8.
Forster A, Brand F, Banan R, Huneburg R, Weber C, Ewert W, et al.
Acta Neuropathol
. 2021 Apr;
142(1):191-210.
PMID: 33929593
The genetic basis of brain tumor development is poorly understood. Here, leukocyte DNA of 21 patients from 15 families with ≥ 2 glioma cases each was analyzed by whole-genome or...
9.
Osmanovic A, Widjaja M, Forster A, Weder J, Wattjes M, Lange I, et al.
J Neurol
. 2020 May;
267(9):2732-2743.
PMID: 32447552
Amyotrophic lateral sclerosis (ALS) and hereditary spastic paraplegia (HSP) are motor neuron diseases sharing clinical, pathological, and genetic similarities. While biallelic SPG7 mutations are known to cause recessively inherited HSP,...
10.
FOCAD loss impacts microtubule assembly, G2/M progression and patient survival in astrocytic gliomas
Brand F, Forster A, Christians A, Bucher M, Thome C, Raab M, et al.
Acta Neuropathol
. 2019 Sep;
139(1):175-192.
PMID: 31473790
In search of novel genes associated with glioma pathogenesis, we have previously shown frequent deletions of the KIAA1797/FOCAD gene in malignant gliomas, and a tumor suppressor function of the encoded...