Alexandra Kavushansky
Overview
Explore the profile of Alexandra Kavushansky including associated specialties, affiliations and a list of published articles.
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14
Citations
354
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Recent Articles
1.
Lichtman D, Bergmann E, Kavushansky A, Cohen N, Levy N, Levy A, et al.
Transl Psychiatry
. 2021 Mar;
11(1):181.
PMID: 33753721
IQSEC2 is an X-linked gene that is associated with autism spectrum disorder (ASD), intellectual disability, and epilepsy. IQSEC2 is a postsynaptic density protein, localized on excitatory synapses as part of...
2.
Bergmann E, Gofman X, Kavushansky A, Kahn I
Commun Biol
. 2020 Dec;
3(1):738.
PMID: 33277621
In recent years precision fMRI has emerged in human brain research, demonstrating characterization of individual differences in brain organization. However, mechanistic investigations to the sources of individual variability are limited...
3.
Asleh J, Shofty B, Cohen N, Kavushansky A, Lopez-Juarez A, Constantini S, et al.
Proc Natl Acad Sci U S A
. 2020 Aug;
117(36):22506-22513.
PMID: 32839340
Neurofibromin gene () mutation causes neurofibromatosis type 1 (NF1), a disorder in which brain white matter deficits identified by neuroimaging are common, yet of unknown cellular etiology. In mice, loss...
4.
Shofty B, Bergmann E, Zur G, Asleh J, Bosak N, Kavushansky A, et al.
Neurobiol Dis
. 2019 May;
130:104479.
PMID: 31128207
Children with the autosomal dominant single gene disorder, neurofibromatosis type 1 (NF1), display multiple structural and functional changes in the central nervous system, resulting in neuropsychological cognitive abnormalities. Here we...
5.
Rogers E, Jada R, Schragenheim-Rozales K, Sah M, Cortes M, Florence M, et al.
Front Mol Neurosci
. 2019 Mar;
12:43.
PMID: 30842726
We have recently described an A350V mutation in IQSEC2 associated with intellectual disability, autism and epilepsy. We sought to understand the molecular pathophysiology of this mutation with the goal of...
6.
Moench K, Maroun M, Kavushansky A, Wellman C
Neurobiol Stress
. 2016 Feb;
3:23-33.
PMID: 26844245
Dysfunction in corticolimbic circuits that mediate the extinction of learned fear responses is thought to underlie the perseveration of fear in stress-related psychopathologies, including post-traumatic stress disorder. Chronic stress produces...
7.
Maroun M, Ioannides P, Bergman K, Kavushansky A, Holmes A, Wellman C
Eur J Neurosci
. 2013 May;
38(4):2611-20.
PMID: 23714419
Stress-sensitive psychopathologies such as post-traumatic stress disorder are characterized by deficits in fear extinction and dysfunction of corticolimbic circuits mediating extinction. Chronic stress facilitates fear conditioning, impairs extinction, and produces...
8.
Asor E, Belhanes H, Kavushansky A, Zubedat S, Klein E, Avital A, et al.
Psychoneuroendocrinology
. 2013 May;
38(10):2173-83.
PMID: 23669323
Background: It is currently accepted that complex behavior and mental disorder results from a combination of biological susceptibility and exposure to environmental stimuli. Most of the gene-environment interaction models focus...
9.
Kavushansky A, Kritman M, Maroun M, Klein E, Richter-Levin G, Hui K, et al.
Eur Neuropsychopharmacol
. 2013 Jan;
23(12):1779-88.
PMID: 23352317
Reactivity to traumatic stress varies between individuals and only a minority of those exposed to trauma develops stress-induced psychopathologies. Currently extensive effort is made to unravel the specific mechanisms predisposing...
10.
Enhanced extinction of aversive memories by high-frequency stimulation of the rat infralimbic cortex
Maroun M, Kavushansky A, Holmes A, Wellman C, Motanis H
PLoS One
. 2012 May;
7(5):e35853.
PMID: 22586453
Electrical stimulation of the rodent medial prefrontal cortex (mPFC), including the infralimbic cortex (IL), immediately prior to or during fear extinction training facilitates extinction memory. Here we examined the effects...