Alejandro Ciocci Pardo
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Explore the profile of Alejandro Ciocci Pardo including associated specialties, affiliations and a list of published articles.
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18
Citations
99
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Recent Articles
1.
Gonzalez Arbelaez L, Ciocci Pardo A, Burgos J, Vila Petroff M, Coto J, Ennis I, et al.
Arch Biochem Biophys
. 2024 Jun;
758:110059.
PMID: 38936683
Background: It has been previously demonstrated that the maintenance of ischemic acidic pH or the delay of intracellular pH recovery at the onset of reperfusion decreases ischemic-induced cardiomyocyte death. Objective:...
2.
Ciocci Pardo A, Diaz Zegarra L, Arbelaez L, Aiello E, Mosca S
Front Pharmacol
. 2023 Aug;
14:1223132.
PMID: 37637427
The increase of intracellular Ca concentration, produced principally by its influx through the L-type Ca channels, is one of the major contributors to the ischemia-reperfusion injury. The inhibition of those...
3.
Ciocci Pardo A, Diaz Zegarra L, Arbelaez L, Ibanez A, Diaz R, Aiello E, et al.
Biochim Biophys Acta Gen Subj
. 2022 Feb;
1866(5):130098.
PMID: 35104623
Our objective was to examine the effects of N-methylacetazolamide (NMA), a non‑carbonic anhydrase inhibitor, on ischemia-reperfusion injury. Isolated rat hearts were assigned to the following groups: 1) Non-ischemic control (NIC):110 ...
4.
Ibanez A, Gonzalez Arbelaez L, Ciocci Pardo A, Mosca S, Lofeudo J, Velez Rueda J, et al.
Biochim Biophys Acta Gen Subj
. 2021 Nov;
1866(2):130060.
PMID: 34822923
During menopause women are exposed to an increase in cardiovascular risk. G protein-coupled estrogen receptor (GPER) is known to mediate several of the protective effects of such hormones. G1 was...
5.
Ciocci Pardo A, Arbelaez L, Fantinelli J, Alvarez B, Mosca S, Swenson E
Physiol Rep
. 2021 Nov;
9(22):e15093.
PMID: 34806317
We have previously demonstrated that inhibition of extracellularly oriented carbonic anhydrase (CA) isoforms protects the myocardium against ischemia-reperfusion injury. In this study, our aim was to assess the possible further...
6.
Sepulveda M, Burgos J, Ciocci Pardo A, Arbelaez L, Mosca S, Vila Petroff M
J Cell Mol Med
. 2021 Jan;
24(17):9627-9637.
PMID: 33460250
Sepsis is associated with cardiac dysfunction, which is at least in part due to cardiomyocyte apoptosis. However, the underlying mechanisms are far from being understood. Using the colon ascendens stent...
7.
Arbelaez L, Ciocci Pardo A, Fantinelli J, Rojano B, Schinella G, Mosca S
Naunyn Schmiedebergs Arch Pharmacol
. 2019 Nov;
393(4):629-638.
PMID: 31776590
Purpose: To determine the actions of isoespintanol (Isoesp) on post-ischemic myocardial and mitochondrial alterations. Methods: Hearts removed from Wistar rats were perfused by 20 min. After this period, the coronary...
8.
Ciocci Pardo A, Arbelaez L, Fantinelli J, Aiello E, Mosca S
Biochem Pharmacol
. 2019 Jan;
161:26-36.
PMID: 30615862
The electrogenic sodium bicarbonate co-transporter isoform 1 (NBCe1) plays an important role in ischemia-reperfusion injury. The cardioprotective action of an antibody directed to the extracellular loop 3 (a-L3) of NBCe1...
9.
Arbelaez L, Ciocci Pardo A, Fantinelli J, Schinella G, Mosca S, Rios J
Food Funct
. 2018 Nov;
9(12):6129-6145.
PMID: 30460963
Myocardial ischemia is the leading cause of death worldwide. Despite better outcomes with early coronary artery reperfusion strategies, morbidity and mortality remain significant. The principal myocardial hallmark of myocardial ischemia...
10.
Arbelaez L, Ciocci Pardo A, Swenson E, Alvarez B, Mosca S, Fantinelli J
Exp Mol Pathol
. 2018 Oct;
105(3):345-351.
PMID: 30308197
Background: Recent studies from our laboratory show the cardioprotective action of benzolamide (BZ, carbonic anhydrase inhibitor) against ischemia-reperfusion injury. However, the mechanisms involved have not been fully elucidated. Objective: To...