Aimee Bastidas-Ponce
Overview
Explore the profile of Aimee Bastidas-Ponce including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
28
Citations
572
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Klein D, Palla G, Lange M, Klein M, Piran Z, Gander M, et al.
Nature
. 2025 Jan;
638(8052):1065-1075.
PMID: 39843746
Single-cell genomic technologies enable the multimodal profiling of millions of cells across temporal and spatial dimensions. However, experimental limitations hinder the comprehensive measurement of cells under native temporal dynamics and...
2.
Akindehin S, Liskiewicz A, Liskiewicz D, Bernecker M, Garcia-Caceres C, Drucker D, et al.
Mol Metab
. 2024 Mar;
83:101915.
PMID: 38492844
Objective: The glucose-dependent insulinotropic polypeptide (GIP) decreases body weight via central GIP receptor (GIPR) signaling, but the underlying mechanisms remain largely unknown. Here, we assessed whether GIP regulates body weight...
3.
Grandl G, Collden G, Feng J, Bhattacharya S, Klingelhuber F, Schomann L, et al.
Nat Metab
. 2024 Feb;
6(3):448-457.
PMID: 38418586
Insulin resistance is an early complication of diet-induced obesity (DIO), potentially leading to hyperglycaemia and hyperinsulinaemia, accompanied by adaptive β cell hypertrophy and development of type 2 diabetes. Insulin not...
4.
Cota P, Caliskan O, Bastidas-Ponce A, Jing C, Jaki J, Saber L, et al.
Mol Metab
. 2023 Dec;
79:101853.
PMID: 38103636
Objective: The consequences of mutations in genes associated with monogenic forms of diabetes on human pancreas development cannot be studied in a time-resolved fashion in vivo. More specifically, if recessive...
5.
Cota P, Saber L, Taskin D, Jing C, Bastidas-Ponce A, Vanheusden M, et al.
Front Endocrinol (Lausanne)
. 2023 Nov;
14:1286590.
PMID: 37955006
Introduction: The molecular programs regulating human pancreatic endocrine cell induction and fate allocation are not well deciphered. Here, we investigated the spatiotemporal expression pattern and the function of the neurogenic...
6.
Liskiewicz A, Khalil A, Liskiewicz D, Novikoff A, Grandl G, Maity-Kumar G, et al.
Nat Metab
. 2023 Nov;
5(12):2075-2085.
PMID: 37946085
The development of single-molecule co-agonists for the glucagon-like peptide-1 (GLP-1) receptor (GLP-1R) and glucose-dependent insulinotropic polypeptide (GIP) receptor (GIPR) is considered a breakthrough in the treatment of obesity and type...
7.
Hrovatin K, Bastidas-Ponce A, Bakhti M, Zappia L, Buttner M, Salinno C, et al.
Nat Metab
. 2023 Sep;
5(9):1615-1637.
PMID: 37697055
Although multiple pancreatic islet single-cell RNA-sequencing (scRNA-seq) datasets have been generated, a consensus on pancreatic cell states in development, homeostasis and diabetes as well as the value of preclinical animal...
8.
Quarta C, Stemmer K, Novikoff A, Yang B, Klingelhuber F, Harger A, et al.
Nat Metab
. 2022 Aug;
4(8):1071-1083.
PMID: 35995995
Dual agonists activating the peroxisome proliferator-activated receptors alpha and gamma (PPARɑ/ɣ) have beneficial effects on glucose and lipid metabolism in patients with type 2 diabetes, but their development was discontinued...
9.
Bakhti M, Bastidas-Ponce A, Tritschler S, Czarnecki O, Tarquis-Medina M, Nedvedova E, et al.
Nat Commun
. 2022 Aug;
13(1):4540.
PMID: 35927244
During pancreas development endocrine cells leave the ductal epithelium to form the islets of Langerhans, but the morphogenetic mechanisms are incompletely understood. Here, we identify the Ca-independent atypical Synaptotagmin-13 (Syt13)...
10.
Tarquis-Medina M, Scheibner K, Gonzalez-Garcia I, Bastidas-Ponce A, Sterr M, Jaki J, et al.
Int J Mol Sci
. 2021 Nov;
22(22).
PMID: 34830411
Synaptotagmin-13 (Syt13) is an atypical member of the vesicle trafficking synaptotagmin protein family. The expression pattern and the biological function of this Ca-independent protein are not well resolved. Here, we...