Adrian Manea
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Explore the profile of Adrian Manea including associated specialties, affiliations and a list of published articles.
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33
Citations
810
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Recent Articles
11.
Manea S, Constantin A, Manda G, Sasson S, Manea A
Redox Biol
. 2015 Jul;
5:358-366.
PMID: 26133261
NADPH oxidases (Nox) represent a family of hetero-oligomeric enzymes whose exclusive biological function is the generation of reactive oxygen species (ROS). Nox-derived ROS are essential modulators of signal transduction pathways...
12.
Manda G, Isvoranu G, Comanescu M, Manea A, Debelec Butuner B, Korkmaz K
Redox Biol
. 2015 Jul;
5:347-357.
PMID: 26122399
The review pinpoints operational concepts related to the redox biology network applied to the pathophysiology and therapeutics of solid tumors. A sophisticated network of intrinsic and extrinsic cues, integrated in...
13.
Manea A, Manea S, Gan A, Constantin A, Fenyo I, Raicu M, et al.
Biochem Biophys Res Commun
. 2015 Apr;
461(1):172-9.
PMID: 25871798
Monocytes (Mon) and Mon-derived macrophages (Mac) orchestrate important oxidative and inflammatory reactions in atherosclerosis by secreting reactive oxygen species (ROS) due, in large part, to the upregulated NADPH oxidases (Nox)....
14.
Manea A, Manea S, Todirita A, Albulescu I, Raicu M, Sasson S, et al.
Cell Tissue Res
. 2015 Feb;
361(2):593-604.
PMID: 25722086
High glucose induces vascular smooth muscle cell (SMC) dysfunction by generating oxidative stress attributable, in part, to the up-regulated NADPH oxidases (Nox). We have attempted to elucidate the high-glucose-generated molecular...
15.
Gan A, Butoi E, Manea A, Pirvulescu M, Stan D, Simion V, et al.
FEBS J
. 2014 Jul;
281(17):3869-81.
PMID: 25040843
Fractalkine (Fk) and its receptor CX3C receptor 1 contribute effectively to the atherosclerotic process, mediating the recruitment of leukocytes and promoting the interactions between monocytes/macrophages and smooth muscle cells (SMCs)....
16.
Manea S, Todirita A, Raicu M, Manea A
J Cell Mol Med
. 2014 May;
18(7):1467-77.
PMID: 24797079
In atherosclerosis, oxidative stress-induced vascular smooth muscle cells (SMCs) dysfunction is partially mediated by up-regulated NADPH oxidase (Nox); the mechanisms of enzyme regulation are not entirely defined. CCAAT/enhancer-binding proteins (C/EBP)...
17.
Manea S, Todirita A, Manea A
PLoS One
. 2013 Dec;
8(12):e84170.
PMID: 24376792
High glucose-induced endothelial dysfunction is partially mediated by the down-stream pathophysiological effects triggered by increased expression of endothelin-1 (ET-1). The molecular control mechanisms of ET-1 synthesis are yet to be...
18.
Manea A, Fortuno A, Martin-Ventura J
Oxid Med Cell Longev
. 2013 Jan;
2012:373450.
PMID: 23346282
No abstract available.
19.
Gan A, Butoi E, Manea A, Simion V, Stan D, Parvulescu M, et al.
Cell Tissue Res
. 2012 Oct;
351(1):161-74.
PMID: 23086480
In the atherosclerotic plaque, smooth muscle cells (SMC) acquire an inflammatory phenotype. Resistin and fractalkine (CX3CL1) are found in human atheroma and not in normal arteries. CX3CL1 and CX3CR1 are...
20.
Manea A, Manea S, Florea I, Luca C, Raicu M
Free Radic Biol Med
. 2012 Feb;
52(9):1497-507.
PMID: 22348975
NADPH oxidase Nox5 subtype expression is significantly increased in vascular smooth muscle cells (SMCs) underlying fibro-lipid atherosclerotic lesions. The mechanisms that up-regulate Nox5 are not understood. Consequently, we characterized the...