» Articles » PMID: 1890216

The Onset Age of Periodontal Destruction

Overview
Date 1991 Jul 1
PMID 1890216
Citations 14
Authors
Affiliations
Soon will be listed here.
Abstract

In the present paper, the onset age of periodontal destruction is discussed. Results of epidemiological studies have shown that with increasing age, the prevalence of periodontitis in a population increases. Data show that after the age of 40 years, the % of the population affected by periodontitis remains rather constant. If it is supposed that the incidence of periodontitis after the age of 40 is zero (no new cases), then extractions resulting in edentulousness due to periodontitis should lead to a decrease of the prevalence of periodontitis. However, after the age of 40 years, the prevalence of periodontitis remains about the same, whereas edentulousness due to periodontitis increases. This suggests that even at a relatively old age, the onset of periodontitis may occur. On the other hand, periodontal destruction has also been found in the primary dentition. Especially on the basis of family studies and studies on localized juvenile periodontitis patients, it can be supposed that in a substantial part of the population, periodontal destruction starts early in life.

Citing Articles

Periodontal diseases in Thai schoolchildren. Clinical and microbiological observations.

Piwat S, Basic A, Pahumunto N, Teanpaisan R, Dahlen G Odontology. 2023; 112(1):232-241.

PMID: 37154987 PMC: 10776494. DOI: 10.1007/s10266-023-00817-w.


Analysis of senescence in gingival tissues and gingival fibroblast cultures.

Furukawa M, Matsuda K, Aoki Y, Yamada M, Wang J, Watanabe M Clin Exp Dent Res. 2022; 8(4):939-949.

PMID: 35491709 PMC: 9382052. DOI: 10.1002/cre2.581.


Association between metabolic syndrome and periodontitis: The role of lipids, inflammatory cytokines, altered host response, and the microbiome.

Pirih F, Monajemzadeh S, Singh N, Sinacola R, Shin J, Chen T Periodontol 2000. 2021; 87(1):50-75.

PMID: 34463996 PMC: 8457155. DOI: 10.1111/prd.12379.


Current understanding of periodontal disease pathogenesis and targets for host-modulation therapy.

Hajishengallis G, Chavakis T, Lambris J Periodontol 2000. 2020; 84(1):14-34.

PMID: 32844416 PMC: 7457922. DOI: 10.1111/prd.12331.


Sucrose and starch intake contribute to reduced alveolar bone height in a rodent model of naturally occurring periodontitis.

Morimoto J, Senior A, Ruiz K, Wali J, Pulpitel T, Solon-Biet S PLoS One. 2019; 14(3):e0212796.

PMID: 30865648 PMC: 6415785. DOI: 10.1371/journal.pone.0212796.