Yukio Takamatsu
Overview
Explore the profile of Yukio Takamatsu including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
25
Citations
519
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Kotajima-Murakami H, Hagihara H, Sato A, Hagino Y, Tanaka M, Katoh Y, et al.
Front Psychiatry
. 2022 Feb;
13:821354.
PMID: 35185658
Autism spectrum disorder (ASD) is a neurodevelopmental disorder that is characterized by impairments in social interaction and restricted/repetitive behaviors. The neurotransmitter γaminobutyric acid (GABA) through GABA receptor signaling in the...
2.
Iwata K, Takamatsu Y, Doi N, Ikeda K
Mol Pain
. 2021 Feb;
17:1744806921992628.
PMID: 33599155
Electroconvulsive therapy (ECT) has been applied for chronic pain for decades. The amounts of opioids to treat pain are sometimes reduced after a series of ECT. The effect of ECT...
3.
Kotajima-Murakami H, Kobayashi T, Kashii H, Sato A, Hagino Y, Tanaka M, et al.
Mol Brain
. 2019 Jan;
12(1):3.
PMID: 30621732
The mammalian target of rapamycin (mTOR) signaling pathway plays a crucial role in cell metabolism, growth, and proliferation. The overactivation of mTOR has been implicated in the pathogenesis of syndromic...
4.
Tanaka M, Sato A, Kasai S, Hagino Y, Kotajima-Murakami H, Kashii H, et al.
Mol Autism
. 2018 Dec;
9:60.
PMID: 30498565
Background: Hyperserotonemia in the brain is suspected to be an endophenotype of autism spectrum disorder (ASD). Reducing serotonin levels in the brain through modulation of serotonin transporter function may improve...
5.
Ide S, Ikekubo Y, Hua J, Takamatsu Y, Uhl G, Sora I, et al.
Neuropsychopharmacol Rep
. 2018 Sep;
38(3):149-153.
PMID: 30175528
Aim: Attention-deficit/hyperactivity disorder is a heterogeneous neurobiological disorder that is characterized by inattention, impulsivity, and an increase in motor activity. Although methylphenidate has been used as a medication for decades,...
6.
Fujita M, Hagino Y, Takamatsu Y, Shimizu Y, Takamatsu Y, Ikeda K, et al.
Neuropsychopharmacol Rep
. 2018 Aug;
38(2):95-97.
PMID: 30106256
Aim: We previously generated transgenic (Tg) mice that expressed P123H β-synuclein (βS), a dementia with Lewy body-linked mutant βS. Notably, these mice recapitulated neurodegenerative features of Lewy body disease, reflected...
7.
Fujita M, Hagino Y, Takeda T, Kasai S, Tanaka M, Takamatsu Y, et al.
Mol Brain
. 2017 Oct;
10(1):49.
PMID: 29037208
Dopamine is important for motor control and involved in the regulation of circadian rhythm. We previously found that dopamine-deficient (DD) mice became hyperactive in a novel environment 72 h after...
8.
Ide S, Takahashi T, Takamatsu Y, Uhl G, Niki H, Sora I, et al.
Int J Neuropsychopharmacol
. 2016 Dec;
20(5):403-409.
PMID: 28031268
Background: Opioid and dopamine systems play crucial roles in reward. Similarities and differences in the neural mechanisms of reward that are mediated by these 2 systems have remained largely unknown....
9.
Yamamoto H, Kamegaya E, Hagino Y, Takamatsu Y, Sawada W, Matsuzawa M, et al.
Neuropharmacology
. 2016 Aug;
112(Pt A):188-197.
PMID: 27480795
The N-methyl-d-aspartate (NMDA) receptor channel is involved in various physiological functions, including learning and memory. The GluN2D subunit of the NMDA receptor has low expression in the mature brain, and...
10.
Yamamoto H, Kamegaya E, Sawada W, Hasegawa R, Yamamoto T, Hagino Y, et al.
Mol Brain
. 2013 Dec;
6:56.
PMID: 24330819
Background: Noncompetitive N-methyl-d-aspartate (NMDA) receptor antagonists evoke a behavioral and neurobiological syndrome in experimental animals. We previously reported that phencyclidine (PCP), an NMDA receptor antagonist, increased locomotor activity in wildtype...