Wenman Wu
Overview
Explore the profile of Wenman Wu including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
51
Citations
226
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Guo P, Wu X, Yang M, Xue Y, Zhou J, Huang Z, et al.
Blood Cells Mol Dis
. 2024 Jul;
109:102874.
PMID: 39032214
Chediak-Higashi syndrome (CHS) is a rare autosomal recessive genetic disorder characterized by severe immunodeficiency, albinism and coagulation deficiency. Mostly diagnosed in early childhood, this devastating condition is associated with lysosomal...
2.
Wu X, Li L, Lu Z, Hu X, Lu Y, Liu Y, et al.
Thromb Haemost
. 2024 Jun;
125(1):69-81.
PMID: 38914130
Background: Venous thromboembolism (VTE) is predisposed by thrombotic mutations in patients with hereditary thrombophilia. Although prothrombin deficiencies caused by homozygous or compound heterozygous mutations are associated with bleeding diathesis, rare...
3.
Mutation Ter462GlnextTer17 introduces a tail to C-terminus of protein C and causes venous thrombosis
Lai Z, Li J, Zhou S, Wu X, Yuan J, Li F, et al.
Thromb Res
. 2024 Jun;
240:109044.
PMID: 38824799
Protein C (PC), a vitamin K-dependent serine protease zymogen in plasma, can be activated by thrombin-thrombomodulin(TM) complex, resulting in the formation of activated protein C (APC). APC functions to downregulate...
4.
Zhou S, Li F, Lai Z, Wu X, Yuan J, Wu W, et al.
J Thromb Haemost
. 2024 May;
22(8):2270-2280.
PMID: 38788977
Background: Protein C (PC) pathway serves as a major defense mechanism against thrombosis by the activation of PC through the thrombin-thrombomodulin complex and subsequent inactivation of the activated factor (F)V...
5.
Liang Q, Zhang Z, Ding B, Shao Y, Ding Q, Dai J, et al.
Thromb Res
. 2024 Feb;
236:51-60.
PMID: 38387303
Introduction: A novel variant involving noncanonical splicing acceptor site (c.875-5 T > G) in propeptide coding region of von Willebrand factor (VWF) was identified in a patient with type 2A...
6.
Zhou S, Wu X, Song Y, Li L, Shi C, Lai Z, et al.
Thromb Haemost
. 2023 Nov;
124(5):459-470.
PMID: 38011863
Background: Protein C (PC) is a vitamin K-dependent anticoagulant serine protease zymogen which upon activation by the thrombin-thrombomodulin (TM) complex downregulates the coagulation cascade by degrading cofactors Va and VIIIa...
7.
Li L, Li J, Wu X, Wu W, Ding Q, Qian B, et al.
Thromb J
. 2023 Oct;
21(1):103.
PMID: 37789321
Background: R189W and K193del of protein C (PC) were hotspot mutations in Chinese population with venous thromboembolism (VTE), but almost two-thirds of patients with above mutations coexisting with other genetically...
8.
Li Y, Liang Q, Wu W, Hu X, Wang H, Wang X, et al.
Res Pract Thromb Haemost
. 2023 Aug;
7(5):102145.
PMID: 37601017
Background: Variants of fibrinogen sequences that bind to thrombin's catalytic sites are mostly associated with bleeding phenotypes, while variants with fibrinogen nonsubstrate-thrombin-binding sites are commonly believed to cause thrombosis. AαGlu39...
9.
Wang R, Jiang S, Wang X, Wei D, Xu Q, Wu W
Int J Hematol
. 2023 May;
118(2):201-209.
PMID: 37210691
Aim: To structurally and functionally characterize three newly identified F9 missense mutations, C268Y, I316F, and G413V, in Chinese hemophilia B patients. Methods: FIX mutants were expressed in vitro by transient...
10.
Li L, Wu X, Wu W, Ding Q, Wang X
Thromb J
. 2023 May;
21(1):52.
PMID: 37143073
Background: Factor VII deficiency is a rare bleeding disorder caused by a deficiency of clotting factor VII. However, there have been some case reports of venous thrombosis in patients with...