Volha Ninichuk
Overview
Explore the profile of Volha Ninichuk including associated specialties, affiliations and a list of published articles.
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Articles
6
Citations
245
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0
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Recent Articles
1.
Ninichuk V, Anders H
Front Biosci
. 2008 May;
13:5163-73.
PMID: 18508578
Bone marrow-derived cells modulate solid organ diseases. For example, the different immune cell populations mediate tissue inflammation and damage, whereas progenitor cell populations are thought to enhance tissue regeneration. However,...
2.
Ninichuk V, Clauss S, Kulkarni O, Schmid H, Segerer S, Radomska E, et al.
Am J Pathol
. 2008 Feb;
172(3):628-37.
PMID: 18258851
Diabetic kidney disease is associated with monocyte chemoattractant CC chemokine ligand 2 (CCL2)-dependent glomerular and interstitial macrophage recruitment. In addition, nephropathy is delayed in Ccl2 mutant diabetic mice. However, whether...
3.
Kulkarni O, Pawar R, Purschke W, Eulberg D, Selve N, Buchner K, et al.
J Am Soc Nephrol
. 2007 Jul;
18(8):2350-8.
PMID: 17625118
The monocyte chemoattractant protein CCL2 is crucial for monocyte and T cell recruitment from the vascular to the extravascular compartment at sites of inflammation. CCL2 is expressed in human lupus...
4.
Ninichuk V, Khandoga A, Segerer S, Loetscher P, Schlapbach A, Revesz L, et al.
Am J Pathol
. 2007 Mar;
170(4):1267-76.
PMID: 17392166
Diabetic nephropathy is associated with interstitial macrophage infiltrates, but their contribution to disease progression is unclear. We addressed this question by blockade of chemokine receptor (CCR)1 because CCR1 mediates the...
5.
Ninichuk V, Anders H
Am J Nephrol
. 2005 Aug;
25(4):365-72.
PMID: 16088077
Infiltrating leukocytes are thought to contribute to the progression of kidney disease. Locally produced chemokines guide circulating leukocytes into the kidney, which renders therapeutic blockade of respective chemokine receptors on...
6.
Ninichuk V, Gross O, Reichel C, Khandoga A, Pawar R, Ciubar R, et al.
J Am Soc Nephrol
. 2005 Feb;
16(4):977-85.
PMID: 15716328
Human Alport disease is caused by a lack of the alpha3-, 4-, or 5-chain of type IV collagen (COL4A). Affected humans and COL4A3-deficient mice develop glomerulosclerosis and progressive renal fibrosis...