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» Authors » Victor B Narkevich

Victor B Narkevich

Explore the profile of Victor B Narkevich including associated specialties, affiliations and a list of published articles. Areas
Snapshot
Articles 6
Citations 37
Followers 0
Related Specialties
Neurology
Psychology
Social Sciences
Top 10 Co-Authors
Vladimir S Kudrin
Chao Wang
Marina A Gruden
Tatiana V Davydova
Ludmilla A Morozova-Roche
Robert D E Sewell
Valentina G Fomina
Carsten T Wotjak
Christoph K Thoeringer
Alexandra V Gabova
Published In
Behav Brain Res
Eur J Pharmacol
Int J Mol Sci
ACS Chem Neurosci
Affiliations
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Recent Articles
1.
Antidepressant and Anxiolytic Effects of L-Methionine in the WAG/Rij Rat Model of Depression Comorbid with Absence Epilepsy
Sarkisova K, Gabova A, Fedosova E, Shatskova A, Narkevich V, Kudrin V
Int J Mol Sci . 2023 Aug; 24(15). PMID: 37569798
Depression is a severe and widespread psychiatric disease that often accompanies epilepsy. Antidepressant treatment of depression comorbid with epilepsy is a major concern due to the risk of seizure aggravation....
2.
S100A9 Protein Aggregates Boost Hippocampal Glutamate Modifying Monoaminergic Neurochemistry: A Glutamate Antibody Sensitive Outcome on Alzheimer-like Memory Decline
Gruden M, Davydova T, Kudrin V, Wang C, Narkevich V, Morozova-Roche L, et al.
ACS Chem Neurosci . 2017 Nov; 9(3):568-577. PMID: 29160692
Alzheimer's disease (AD) involves dementia conceivably arising from integrated inflammatory processes, amyloidogenesis, and neuronal apoptosis. Glutamate can also cause neuronal death via excitotoxicity, and this is similarly implicated in some...
3.
The misfolded pro-inflammatory protein S100A9 disrupts memory via neurochemical remodelling instigating an Alzheimer's disease-like cognitive deficit
Gruden M, Davydova T, Wang C, Narkevich V, Fomina V, Kudrin V, et al.
Behav Brain Res . 2016 Mar; 306:106-16. PMID: 26965570
Memory deficits may develop from a variety of neuropathologies including Alzheimer's disease dementia. During neurodegenerative conditions there are contributory factors such as neuroinflammation and amyloidogenesis involved in memory impairment. In...
4.
Noradrenergic and serotonergic neurochemistry arising from intranasal inoculation with α-synuclein aggregates which incite parkinsonian-like symptoms
Gruden M, Davydova T, Narkevich V, Fomina V, Wang C, Kudrin V, et al.
Behav Brain Res . 2014 Dec; 279:191-201. PMID: 25446742
Alpha-synuclein (α-syn) toxic aggregates delivered by the nasal vector have been shown to modify the neurochemistry of dopamine (DA) which is associated with parkinsonian-like motor symptoms. The aim was therefore...
5.
Intranasal administration of alpha-synuclein aggregates: a Parkinson's disease model with behavioral and neurochemical correlates
Gruden M, Davydova T, Narkevich V, Fomina V, Wang C, Kudrin V, et al.
Behav Brain Res . 2014 Feb; 263:158-68. PMID: 24480422
Parkinson's disease (PD) is a neurodegenerative disorder in which both alpha-synuclein (α-syn) and dopamine (DA) have a critical role. Our previous studies instigated a novel PD model based on nasal...
6.
Strain differences in profiles of dopaminergic neurotransmission in the prefrontal cortex of the BALB/C vs. C57Bl/6 mice: consequences of stress and afobazole
Anderzhanova E, Bachli H, Buneeva O, Narkevich V, Medvedev A, Thoeringer C, et al.
Eur J Pharmacol . 2013 Mar; 708(1-3):95-104. PMID: 23524098
We found that in mice the basal activity of monoamine oxidase B (MAO-B) in the medial prefrontal cortex (mPFC) is lower in BALB/C than in C57Bl/6J mice, whereas activity of...