Vera Dobelmann
Overview
Explore the profile of Vera Dobelmann including associated specialties, affiliations and a list of published articles.
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12
Citations
36
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Recent Articles
1.
Malaichamy S, Idoux R, Polavarapu K, Sikic K, Holla E, Thompson R, et al.
Brain
. 2025 Feb;
PMID: 39970126
Rhabdomyolysis is an acute failure of cellular homeostasis resulting in muscle breakdown, triggered by trauma, infection, drugs, or strenuous exercise. Recurrent rhabdomyolysis is often associated with genetic and metabolic defects...
2.
Dobelmann V, Roos A, Hentschel A, Marina A, Leo M, Schmitt L, et al.
J Neurol
. 2024 Sep;
271(10):7000-7011.
PMID: 39240344
Background And Purpose: Spinal muscular atrophy (SMA) as the second most common neurodegenerative disorder in childhood is characterized by the deficiency of survival of motor neuron (SMN) protein leading predominantly...
3.
Nelke C, Schroeter C, Barman S, Stascheit F, Masanneck L, Theissen L, et al.
EBioMedicine
. 2024 Jul;
105:105231.
PMID: 38959848
Background: The clinical heterogeneity of myasthenia gravis (MG), an autoimmune disease defined by antibodies (Ab) directed against the postsynaptic membrane, constitutes a challenge for patient stratification and treatment decision making....
4.
Schroeter C, Nelke C, Stascheit F, Huntemann N, Preusse C, Dobelmann V, et al.
Acta Neuropathol
. 2024 Jun;
147(1):102.
PMID: 38888758
Myasthenia gravis is a chronic antibody-mediated autoimmune disease disrupting neuromuscular synaptic transmission. Informative biomarkers remain an unmet need to stratify patients with active disease requiring intensified monitoring and therapy; their...
5.
Theissen L, Schroeter C, Huntemann N, Rauber S, Dobelmann V, Cengiz D, et al.
Cells
. 2024 Mar;
13(6.
PMID: 38534352
Myasthenia gravis (MG) is a prototypical autoimmune disease of the neuromuscular junction (NMJ). The study of the underlying pathophysiology has provided novel insights into the interplay of autoantibodies and complement-mediated...
6.
Nelke C, Muntefering T, Cengiz D, Theissen L, Dobelmann V, Schroeter C, et al.
J Autoimmun
. 2023 Nov;
142:103136.
PMID: 37935063
K2.1 (TREK1), a two-pore domain potassium channel, has emerged as regulator of leukocyte transmigration into the central nervous system. In the context of skeletal muscle, immune cell infiltration constitutes the...
7.
Schroeter C, Henes A, Vogelsang A, Herrmann A, Lichtenberg S, Cengiz D, et al.
J Vis Exp
. 2023 Oct;
(200).
PMID: 37870321
Experimental autoimmune encephalomyelitis (EAE) is the most common murine model for multiple sclerosis (MS) and is frequently used to further elucidate the still unknown etiology of MS in order to...
8.
Senescent fibro-adipogenic progenitors are potential drivers of pathology in inclusion body myositis
Nelke C, Schroeter C, Theissen L, Preusse C, Pawlitzki M, Rauber S, et al.
Acta Neuropathol
. 2023 Sep;
146(5):725-745.
PMID: 37773216
Inclusion body myositis (IBM) is unique across the spectrum of idiopathic inflammatory myopathies (IIM) due to its distinct clinical presentation and refractoriness to current treatment approaches. One explanation for this...
9.
Nguyen C, Jimenez-Moreno A, Merker M, Bowers C, Nikolenko N, Hentschel A, et al.
J Neurol
. 2023 Mar;
270(6):3138-3158.
PMID: 36892629
Background And Purpose: Myotonic dystrophy type 1 (DM1) is the most common form of adult-onset muscular dystrophy and is caused by an repeat expansion [r(CUG)] located in the 3' untranslated...
10.
Schroeter C, Rolfes L, Gothan K, Gruchot J, Herrmann A, Bock S, et al.
J Neuroinflammation
. 2022 Nov;
19(1):270.
PMID: 36348455
Background: Cladribine is a synthetic purine analogue that interferes with DNA synthesis and repair next to disrupting cellular proliferation in actively dividing lymphocytes. The compound is approved for the treatment...