Thomas Hackenbeck
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Explore the profile of Thomas Hackenbeck including associated specialties, affiliations and a list of published articles.
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Articles
14
Citations
333
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0
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Recent Articles
1.
Bartel A, Grau J, Bitzegeio J, Werber D, Linzner N, Schumacher V, et al.
Viruses
. 2024 Jan;
16(1).
PMID: 38257802
The importance of COVID-19 surveillance from wastewater continues to grow since case-based surveillance in the general population has been scaled back world-wide. In Berlin, Germany, quantitative and genomic wastewater monitoring...
2.
Schumann V, de Castro Cuadrat R, Wyler E, Wurmus R, Deter A, Quedenau C, et al.
Sci Total Environ
. 2022 Oct;
853:158931.
PMID: 36228784
The use of RNA sequencing from wastewater samples is a valuable way for estimating infection dynamics and circulating lineages of SARS-CoV-2. This approach is independent from testing individuals and can...
3.
Knaup K, Hackenbeck T, Popp B, Stoeckert J, Wenzel A, Buttner-Herold M, et al.
J Am Soc Nephrol
. 2018 Jul;
29(9):2298-2309.
PMID: 30049680
Background: Providing the correct diagnosis for patients with tubulointerstitial kidney disease and secondary degenerative disorders, such as hypertension, remains a challenge. The autosomal dominant tubulointerstitial kidney disease (ADTKD) subtype caused...
4.
Schley G, Scholz H, Kraus A, Hackenbeck T, Klanke B, Willam C, et al.
Kidney Int
. 2015 Jul;
88(6):1283-1292.
PMID: 26200943
Reduced nephron number predisposes to hypertension and kidney disease. Interaction of the branching ureteric bud and surrounding mesenchymal cells determines nephron number. Since oxygen supply may be critical for intrauterine...
5.
Ekici A, Hackenbeck T, Moriniere V, Pannes A, Buettner M, Uebe S, et al.
Kidney Int
. 2014 Mar;
86(3):589-99.
PMID: 24670410
For decades, ill-defined autosomal dominant renal diseases have been reported, which originate from tubular cells and lead to tubular atrophy and interstitial fibrosis. These diseases are clinically indistinguishable, but caused...
6.
Jobst-Schwan T, Knaup K, Nielsen R, Hackenbeck T, Buettner-Herold M, Lechler P, et al.
Am J Physiol Renal Physiol
. 2013 Jul;
305(5):F734-44.
PMID: 23825075
The inhibitor of apoptosis protein survivin is a bifunctional molecule that regulates cellular division and survival. We have previously shown that survivin protein can be found at high concentrations in...
7.
Knaup K, Monti J, Hackenbeck T, Jobst-Schwan T, Klanke B, Schietke R, et al.
Mol Carcinog
. 2013 Jul;
53(12):970-8.
PMID: 23818324
Hypoxia leads to the upregulation of a variety of genes mediated largely via the hypoxia inducible transcription factor (HIF). Prominent HIF-regulated target genes such as the vascular endothelial growth factor...
8.
Schietke R, Hackenbeck T, Tran M, Gunther R, Klanke B, Warnecke C, et al.
PLoS One
. 2012 Feb;
7(1):e31034.
PMID: 22299048
The Hypoxia-inducible transcription Factor (HIF) represents an important adaptive mechanism under hypoxia, whereas sustained activation may also have deleterious effects. HIF activity is determined by the oxygen regulated α-subunits HIF-1α...
9.
Hackenbeck T, Huber R, Schietke R, Knaup K, Monti J, Wu X, et al.
Biochim Biophys Acta
. 2010 Nov;
1813(1):1-13.
PMID: 21056597
Hypoxia is a common pathogenic stress, which requires adaptive activation of the Hypoxia-inducible transcription factor (HIF). In concert transcriptional HIF targets enhance oxygen availability and simultaneously reduce oxygen demand, enabling...
10.
Gimm T, Wiese M, Teschemacher B, Deggerich A, Schodel J, Knaup K, et al.
FASEB J
. 2010 Jul;
24(11):4443-58.
PMID: 20624928
Hypoxia-inducible protein 2 (HIG2) has been implicated in canonical Wnt signaling, both as target and activator. The potential link between hypoxia and an oncogenic signaling pathway might play a pivotal...