Thibaut de Sablet
Overview
Explore the profile of Thibaut de Sablet including associated specialties, affiliations and a list of published articles.
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Articles
24
Citations
1012
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Recent Articles
1.
de Sablet T, Potiron L, Marquis M, Bussiere F, Lacroix-Lamande S, Laurent F
Cell Microbiol
. 2016 Jun;
18(12):1871-1880.
PMID: 27324279
Intestinal epithelial cells form a single layer separating the intestinal lumen containing nutriments and microbiota from the underlying sterile tissue and therefore play a key role in maintaining homeostasis. We...
2.
Bussiere F, Brossier F, Le Vern Y, Niepceron A, Silvestre A, de Sablet T, et al.
PLoS One
. 2015 Feb;
10(2):e0116509.
PMID: 25689363
E. tenella infection is associated with a severe intestinal disease leading to high economic losses in poultry industry. Mitogen activated protein kinases (MAPKs) are implicated in early response to infection...
3.
Heme oxygenase-1 dysregulates macrophage polarization and the immune response to Helicobacter pylori
Gobert A, Verriere T, Asim M, Barry D, Piazuelo M, de Sablet T, et al.
J Immunol
. 2014 Aug;
193(6):3013-22.
PMID: 25108023
Helicobacter pylori incites a futile inflammatory response, which is the key feature of its immunopathogenesis. This leads to the ability of this bacterial pathogen to survive in the stomach and...
4.
Kodaman N, Pazos A, Schneider B, Piazuelo M, Mera R, Sobota R, et al.
Proc Natl Acad Sci U S A
. 2014 Jan;
111(4):1455-60.
PMID: 24474772
Helicobacter pylori is the principal cause of gastric cancer, the second leading cause of cancer mortality worldwide. However, H. pylori prevalence generally does not predict cancer incidence. To determine whether...
5.
Schneider B, Piazuelo M, Sicinschi L, Mera R, Peng D, Roa J, et al.
Epigenetics
. 2013 Oct;
8(11):1153-61.
PMID: 24128875
DNA methylation changes are known to occur in gastric cancers and in premalignant lesions of the gastric mucosae. In order to examine variables associated with methylation levels, we quantitatively evaluated...
6.
Hardbower D, de Sablet T, Chaturvedi R, Wilson K
Gut Microbes
. 2013 Jul;
4(6):475-81.
PMID: 23811829
Helicobacter pylori is the leading risk factor associated with gastric carcinogenesis. H. pylori leads to chronic inflammation because of the failure of the host to eradicate the infection. Chronic inflammation...
7.
Gobert A, Verriere T, de Sablet T, Peek Jr R, Chaturvedi R, Wilson K
Cell Microbiol
. 2012 Oct;
15(1):145-56.
PMID: 23051580
The cytotoxin-associated gene A protein (CagA) plays a pivotal role in the aetiology of Helicobacter pylori-associated gastric diseases. CagA is injected into the cytoplasm of host cells by a type...
8.
Chaturvedi R, de Sablet T, Peek R, Wilson K
Gut Microbes
. 2012 May;
3(1):48-56.
PMID: 22555547
We have recently reported that Helicobacter pylori strains expressing the virulence factor cytotoxin-associated gene A (CagA) stimulate increased levels of spermine oxidase (SMO) in gastric epithelial cells, while cagA⁻ strains...
9.
Barry D, Asim M, Scull B, Piazuelo M, de Sablet T, Lewis N, et al.
PLoS One
. 2011 Dec;
6(12):e29046.
PMID: 22194986
Once acquired, Helicobacter pylori infection is lifelong due to an inadequate innate and adaptive immune response. Our previous studies indicate that interactions among the various pathways of arginine metabolism in...
10.
Gobert A, Asim M, Piazuelo M, Verriere T, Scull B, de Sablet T, et al.
J Immunol
. 2011 Oct;
187(10):5370-9.
PMID: 21987660
A strong cellular cross-talk exists between the pathogen Helicobacter pylori and high-output NO production. However, how NO and H. pylori interact to signal in gastric epithelial cells and modulate the...