Taiji Matsusaka
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Explore the profile of Taiji Matsusaka including associated specialties, affiliations and a list of published articles.
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94
Citations
2689
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Recent Articles
1.
Okabe M, Okabayashi Y, Sasaki T, Koike K, Tsuboi N, Matsusaka T, et al.
Kidney360
. 2024 Dec;
PMID: 39714942
Background: Lupus nephritis (LN) is a major complication of systemic lupus erythematosus. Like other types of glomerulonephritis, podocyte injury has been observed in patients with LN. However, the association between...
2.
Suzuki Y, Kaseda R, Nakagawa Y, Watanabe H, Otsuka T, Yamamoto S, et al.
Clin Exp Nephrol
. 2024 Dec;
PMID: 39666151
Background: Massive proteinuria, dyslipidemia, and hypoalbuminemia induced by nephrotic syndrome (NS) secondarily affect tubular cells. We conducted an RNA sequencing (RNA-seq) analysis using a mouse model of focal segmental glomerulosclerosis...
3.
Takahashi M, Yamamoto S, Yamamoto S, Okubo A, Nakagawa Y, Kuwahara K, et al.
Nat Commun
. 2024 Nov;
15(1):9977.
PMID: 39578451
Acute kidney injury (AKI), typically caused by ischemia, is a common clinical complication with a poor prognosis. Although proteinuria is an important prognostic indicator of AKI, the underlying causal mechanism...
4.
Tanaka K, Hayasaka H, Matsusaka T
PLoS One
. 2024 May;
19(5):e0303910.
PMID: 38805434
Dach1 is highly expressed in normal podocytes, but this expression rapidly disappears after podocyte injury. To investigate the role of Dach1 in podocytes in vivo, we analyzed global, podocyte-specific, and...
5.
Okabe M, Koike K, Yamamoto I, Tsuboi N, Matsusaka T, Yokoo T
Clin Kidney J
. 2024 Jan;
17(1):sfad289.
PMID: 38186896
Background: In human glomerular diseases, visualizing podocyte injury is desirable since podocytes do not regenerate and podocyte injury leads to podocyte loss. Herein, we investigated the utility of immunostaining for...
6.
Takahashi N, Yokoi S, Kimura H, Naiki H, Matsusaka T, Yamamoto Y, et al.
Sci Rep
. 2023 Dec;
13(1):22540.
PMID: 38110482
Podocyte expression of fibroblast specific protein 1 (FSP1) is observed in various types of human glomerulonephritis. Considering that FSP1 is secreted extracellularly and has been shown to have multiple biological...
7.
Sugioka S, Yamada H, Ishii A, Kato Y, Yamada R, Mori K, et al.
Kidney Int
. 2023 Jun;
104(3):508-525.
PMID: 37356621
Natriuretic peptides exert not only blood-lowering but also kidney-protective effects through guanylyl cyclase-A (GC-A), a natriuretic peptide receptor. Signaling through GC-A has been shown to protect podocytes from aldosterone-induced glomerular...
8.
Nakamura J, Yamamoto T, Takabatake Y, Namba-Hamano T, Minami S, Takahashi A, et al.
JCI Insight
. 2023 Jan;
8(4).
PMID: 36649084
Obesity is a major risk factor for end-stage kidney disease. We previously found that lysosomal dysfunction and impaired autophagic flux contribute to lipotoxicity in obesity-related kidney disease, in both humans...
9.
Tanaka K, Tanaka M, Watanabe N, Ito M, Pastan I, Koizumi M, et al.
Sci Rep
. 2022 Dec;
12(1):22356.
PMID: 36572741
Podoplanin (PDPN) is intensely expressed on the podocyte membrane in an evolutionally conserved manner. CLEC-2, the endogenous ligand of PDPN, is highly expressed in platelets and also exists in a...
10.
Zhong J, Yang H, Shelton E, Matsusaka T, Clark A, Yermalitsky V, et al.
JCI Insight
. 2022 Sep;
7(21).
PMID: 36125905
Lipoprotein modification by reactive dicarbonyls, including isolevuglandin (IsoLG), produces dysfunctional particles. Kidneys participate in lipoprotein metabolism, including tubular uptake. However, the process beyond the proximal tubule is unclear, as is...