T Halttunen
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Explore the profile of T Halttunen including associated specialties, affiliations and a list of published articles.
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14
Citations
469
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Recent Articles
1.
Halttunen T, Collado M, El-Nezami H, Meriluoto J, Salminen S
Lett Appl Microbiol
. 2007 Nov;
46(2):160-5.
PMID: 18028332
Aims: The primary objective of this study was to compare the removal of cadmium, lead, aflatoxin B1 and microcystin-LR from aqueous solution by Lactobacillus rhamnosus GG, L. rhamnosus LC705, Propionibacterium...
2.
Halttunen T, Salminen S, Tahvonen R
Int J Food Microbiol
. 2006 Dec;
114(1):30-5.
PMID: 17184867
Cadmium and lead are highly toxic metals. People are exposed to them primarily through food and water. Available conventional methods (precipitation, flocculation, ion exchange, and membrane filtration) for removal of...
3.
Korponay-Szabo I, Halttunen T, Szalai Z, Laurila K, Kiraly R, Kovacs J, et al.
Gut
. 2004 Apr;
53(5):641-8.
PMID: 15082580
Background: IgA class serum autoantibodies against type 2 (tissue) transglutaminase (TG2) bind to both intestinal and extraintestinal normal tissue sections in vitro, eliciting endomysial, reticulin, and jejunal antibody reactions. It...
4.
Korponay-Szabo I, Laurila K, Szondy Z, Halttunen T, Szalai Z, Dahlbom I, et al.
Gut
. 2003 Jan;
52(2):199-204.
PMID: 12524400
Background: Autoantibodies against transglutaminase 2 (TG2) are thought to be responsible for the endomysial (EMA), reticulin (ARA), and jejunal antibody (JEA) tissue binding of serum samples from coeliac patients but...
5.
Kainulainen H, Rantala I, Collin P, Ruuska T, Paivarinne H, Halttunen T, et al.
Gut
. 2002 Jan;
50(1):84-9.
PMID: 11772972
Background And Aims: Coeliac disease is characterised by atrophy of the villi and hyperplasia of the crypts in the mucosa of the small intestine. It is caused by an environmental...
6.
Lindfors K, Halttunen T, Kainulainen H, Maki M
Life Sci
. 2001 Sep;
69(12):1363-72.
PMID: 11531160
We have previously shown that transforming growth factor-beta1 (TGF-beta1) is involved in the fibroblast-induced organization and differentiation of transformed phenotypically crypt-like T84 intestinal epithelial cells into absorptive enterocyte-like cells, when...
7.
Korponay-Szabo I, Sulkanen S, Halttunen T, Maurano F, Rossi M, Mazzarella G, et al.
J Pediatr Gastroenterol Nutr
. 2001 Jan;
31(5):520-7.
PMID: 11144437
Background: Endomysial antibodies have recently been shown to react with tissue transglutaminase. This study was undertaken to investigate whether the tissue distribution of transglutaminase is also compatible with reticulin, jejunal,...
8.
Lindfors K, Halttunen T, Huotari P, Nupponen N, Vihinen M, Visakorpi T, et al.
Biochem Biophys Res Commun
. 2000 Oct;
276(2):660-6.
PMID: 11027528
Intestinal crypt epithelial T84 cells form luminal structures and differentiate to intestinal enterocyte-like cells in response to IMR-90 fibroblast-secreted transforming growth factor-beta when grown within three-dimensional collagen gel. In search...
9.
Laitinen M, Halttunen T, Jortikka L, Teronen O, Sorsa T, Lindholm T
Life Sci
. 1999 Mar;
64(10):847-58.
PMID: 10096435
Various matrix growth factors play important roles in the development and growth of cartilage and bone. Among them transforming growth factor-beta superfamily and especially bone morphogenetic proteins are known to...
10.
Halttunen T, Maki M
Gastroenterology
. 1999 Feb;
116(3):566-72.
PMID: 10029615
Background & Aims: Celiac disease is characterized by disturbed jejunal crypt-villus axis biology with immunoglobulin (Ig) A deposits underlining the epithelium. The aim of this study was to test whether...