Srikant Rangaraju
Overview
Explore the profile of Srikant Rangaraju including associated specialties, affiliations and a list of published articles.
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90
Citations
2177
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Recent Articles
1.
Wang J, Zhang H, Augenreich M, Martinez-Lemus A L, Liu Z, Kang X, et al.
J Neurochem
. 2025 Feb;
169(3):e70024.
PMID: 40019120
Chemotherapy-related cognitive impairment (CRCI) significantly impacts cancer survivors. Due to unclear mechanisms, effective treatments for cognitive deficits are lacking. Here, we examined if microglia-mediated deficits in synaptic plasticity drive CRCI....
2.
Ramelow C, Dammer E, Xiao H, Cheng L, Kumar P, Espinosa-Garcia C, et al.
bioRxiv
. 2025 Feb;
PMID: 39974879
Deep molecular phenotyping of cells at transcriptomic and proteomic levels is an essential first step to understanding cellular contributions to development, aging, injury, and disease. Since proteome and transcriptome level...
3.
Shapley S, Shantaraman A, Kearney M, Dammer E, Duong D, Bowen C, et al.
bioRxiv
. 2025 Feb;
PMID: 39896523
In Alzheimer's disease (AD) and other tauopathies, tau dissociates from microtubules and forms toxic aggregates that contribute to neurodegeneration. Although some of the pathological interactions of tau have been identified...
4.
Kour D, Bowen C, Srivastava U, Nguyen H, Kumari R, Kumar P, et al.
bioRxiv
. 2025 Jan;
PMID: 39868101
Potassium channels regulate membrane potential, calcium flux, cellular activation and effector functions of adaptive and innate immune cells. The voltage-activated Kv1.3 channel is an important regulator of T cell-mediated autoimmunity...
5.
Entorhinal cortex vulnerability to human APP expression promotes hyperexcitability and tau pathology
Goettemoeller A, Banks E, Kumar P, Olah V, McCann K, South K, et al.
Nat Commun
. 2024 Sep;
15(1):7918.
PMID: 39256379
Preventative treatment for Alzheimer's Disease (AD) is dire, yet mechanisms underlying early regional vulnerability remain unknown. In AD, one of the earliest pathophysiological correlates to cognitive decline is hyperexcitability, which...
6.
Levites Y, Dammer E, Ran Y, Tsering W, Duong D, Abreha M, et al.
Cell Rep Med
. 2024 Aug;
5(8):101669.
PMID: 39127040
Alzheimer's disease (AD) is a complex neurodegenerative disorder that develops over decades. AD brain proteomics reveals vast alterations in protein levels and numerous altered biologic pathways. Here, we compare AD...
7.
Entorhinal cortex vulnerability to human APP expression promotes hyperexcitability and tau pathology
Goettemoeller A, Banks E, Kumar P, Olah V, McCann K, South K, et al.
bioRxiv
. 2024 Jul;
PMID: 39005389
Preventative treatment for Alzheimer's Disease is of dire importance, and yet, cellular mechanisms underlying early regional vulnerability in Alzheimer's Disease remain unknown. In human patients with Alzheimer's Disease, one of...
8.
Bowen C, Nguyen H, Lin Y, Bagchi P, Natu A, Espinosa-Garcia C, et al.
Mol Cell Proteomics
. 2024 Jun;
23(8):100809.
PMID: 38936775
Microglia are resident immune cells of the brain and regulate its inflammatory state. In neurodegenerative diseases, microglia transition from a homeostatic state to a state referred to as disease-associated microglia...
9.
Pybus A, Bitarafan S, Brothers R, Rohrer A, Khaitan A, Moctezuma F, et al.
J Neuroinflammation
. 2024 Jun;
21(1):156.
PMID: 38872143
Repetitive mild traumatic brain injuries (rmTBI) sustained within a window of vulnerability can result in long term cognitive deficits, depression, and eventual neurodegeneration associated with tau pathology, amyloid beta (Aβ)...
10.
Tarek M, Monteiro M, Mohammaden M, Martins P, Sheth S, Dolia J, et al.
J Neurointerv Surg
. 2024 May;
PMID: 38782568
Background: Early identification of intracranial atherosclerotic disease (ICAD) may impact the management of patients undergoing mechanical thrombectomy (MT). We sought to develop and validate a scoring system for pre-thrombectomy diagnosis...