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Sonia Aydin

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Articles 6
Citations 145
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Recent Articles
1.
Wilmes A, Crean D, Aydin S, Pfaller W, Jennings P, Leonard M
Toxicol In Vitro . 2010 Dec; 25(3):613-22. PMID: 21172416
The identification and dissection of cellular stress mechanisms is fundamental to understanding the susceptibility of the kidney to chemicals and pharmaceuticals and for the development of renal biomarkers indicative of...
2.
Jennings P, Aydin S, Bennett J, McBride R, Weiland C, Tuite N, et al.
Toxicol In Vitro . 2009 Jan; 23(3):486-99. PMID: 19159671
There is an acknowledged need to promote and further develop in vitro techniques in order to achieve the goal of improved risk assessment of chemicals and pharmaceuticals to humans. The...
3.
Aydin S, Signorelli S, Lechleitner T, Joannidis M, Pleban C, Perco P, et al.
Am J Physiol Cell Physiol . 2007 Dec; 294(2):C543-54. PMID: 18057119
In the renal cortex the peritubular capillary network and the proximal tubular epithelium cooperate in solute and water reabsorption, secretion, and inflammation. However, the mechanisms by which these two cell...
4.
Jennings P, Koppelstaetter C, Aydin S, Abberger T, Wolf A, Mayer G, et al.
Am J Physiol Renal Physiol . 2007 Jun; 293(3):F831-8. PMID: 17596534
The nephrotoxic potential of the widely used immunosuppressive agent cyclosporine A (CsA) is well recognized. However, the mechanism of renal tubular toxicity is not yet fully elucidated. Chronic CsA nephropathy...
5.
Bijuklic K, Jennings P, Kountchev J, Hasslacher J, Aydin S, Sturn D, et al.
Am J Physiol Cell Physiol . 2007 Apr; 293(1):C486-92. PMID: 17428840
Interstitial inflammation has emerged as a key event in the development of acute renal failure. To gain better insight into the nature of these inflammatory processes, the interplay between tubular...
6.
Jennings P, Aydin S, Kotanko P, Lechner J, Lhotta K, Williams S, et al.
J Am Soc Nephrol . 2006 Dec; 18(1):264-73. PMID: 17151335
Familial juvenile hyperuricemic nephropathy (FJHN) is an autosomal dominant genetic disorder that is characterized by hyperuricemia, gout, and tubulointerstitial nephritis. FJHN is caused by mutations in the UMOD gene, which...