Simona Allocca
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Explore the profile of Simona Allocca including associated specialties, affiliations and a list of published articles.
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10
Citations
56
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Recent Articles
1.
Troise F, Leoni G, Sasso E, Del Sorbo M, Esposito M, Romano G, et al.
Mol Ther Oncol
. 2024 Apr;
32(1):200760.
PMID: 38596303
Neoantigen (neoAg)-based cancer vaccines expand preexisting antitumor immunity and elicit novel cancer-specific T cells. However, at odds with prophylactic vaccines, therapeutic antitumor immunity must be induced when the tumor is...
2.
Bartolomeo R, Troise F, Allocca S, Sdruscia G, Vitale R, Bignone V, et al.
Vaccines (Basel)
. 2024 Mar;
12(3).
PMID: 38543959
Quality control testing of vaccines, including potency assessment, is critical to ensure equivalence of clinical lots. We developed a potency assay to support the clinical advancement of Nous-209, a cancer...
3.
Garzia I, Nocchi L, Avalle L, Troise F, Leoni G, Secli L, et al.
Cancer Immunol Res
. 2024 Feb;
12(4):440-452.
PMID: 38331413
Tumor neoantigens (nAg) represent a promising target for cancer immunotherapy. The identification of nAgs that can generate T-cell responses and have therapeutic activity has been challenging. Here, we sought to...
4.
DAlise A, Nocchi L, Garzia I, Secli L, Infante L, Troise F, et al.
Front Immunol
. 2023 May;
14:1156714.
PMID: 37180141
Introduction: Virus vectored genetic vaccines (Vvgv) represent a promising approach for eliciting immune protection against infectious diseases and cancer. However, at variance with classical vaccines to date, no adjuvant has...
5.
Secli L, Infante L, Nocchi L, De Lucia M, Cotugno G, Leoni G, et al.
J Immunother Cancer
. 2023 Apr;
11(4).
PMID: 37117006
Background: Tumor microenvironment (TME) represents a critical hurdle in cancer immunotherapy, given its ability to suppress antitumor immunity. Several efforts are made to overcome this hostile TME with the development...
6.
Leoni G, DAlise A, Tucci F, Micarelli E, Garzia I, De Lucia M, et al.
Vaccines (Basel)
. 2021 Aug;
9(8).
PMID: 34452005
Neoantigens are tumor-specific antigens able to induce T-cell responses, generated by mutations in protein-coding regions of expressed genes. Previous studies demonstrated that only a limited subset of mutations generates neoantigens...
7.
Parisi S, Polishchuk E, Allocca S, Ciano M, Musto A, Gallo M, et al.
Sci Rep
. 2020 Jun;
10(1):10476.
PMID: 32572046
An amendment to this paper has been published and can be accessed via a link at the top of the paper.
8.
Allocca S, Ciano M, Ciardulli M, DAmbrosio C, Scaloni A, Sarnataro D, et al.
Int J Mol Sci
. 2018 Jun;
19(7).
PMID: 29954118
The H1069Q substitution is the most frequent mutation of the Cu transporter ATP7B that causes Wilson disease in the Caucasian population. ATP7B localizes to the Golgi complex in hepatocytes, but,...
9.
Parisi S, Polishchuk E, Allocca S, Ciano M, Musto A, Gallo M, et al.
Sci Rep
. 2018 Apr;
8(1):6247.
PMID: 29674751
H1069Q substitution represents the most frequent mutation of the copper transporter ATP7B causing Wilson disease in Caucasian population. ATP7B localizes to the Golgi complex in hepatocytes but moves in response...
10.
Ciano M, Allocca S, Ciardulli M, Della Volpe L, Bonatti S, DAgostino M
Biochem Biophys Res Commun
. 2016 Sep;
479(2):325-330.
PMID: 27641668
We have previously shown that αB-crystallin (CRYAB), a small heat shock protein (sHsp) that prevents irreversible aggregation of unfolded protein by an ATP-independent chaperone activity, plays a pivotal role in...