Simon Sherman
Overview
Explore the profile of Simon Sherman including associated specialties, affiliations and a list of published articles.
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1744
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Recent Articles
1.
Downs B, Sherman S, Cui J, Kim Y, Snyder C, Christensen M, et al.
Eur J Cancer
. 2018 Dec;
107:68-78.
PMID: 30551077
Purpose: The presence of pathogenic germline mutation in BRCA1 gene is considered as the most penetrant genetic predisposition for breast cancer. However, a portion of BRCA1 mutation carriers never develops...
2.
Shats O, Goldner W, Feng J, Sherman A, Smith R, Sherman S
Cancer Inform
. 2016 May;
15:73-9.
PMID: 27168721
A multicenter, web-based Thyroid Cancer and Tumor Collaborative Registry (TCCR, http://tccr.unmc.edu) allows for the collection and management of various data on thyroid cancer (TC) and thyroid nodule (TN) patients. The...
3.
Sherman S, Rathnayake N, Mdzinarishvili T
PLoS One
. 2015 Oct;
10(10):e0140405.
PMID: 26465159
Carcinogenic modeling is aimed at mathematical descriptions of cancer development in aging. In this work, we assumed that a small fraction of individuals in the population is susceptible to cancer,...
4.
Downs B, Kim Y, Xiao F, Snyder C, Chen P, Fleissner E, et al.
Breast Cancer Res Treat
. 2015 Apr;
151(1):219-24.
PMID: 25833210
Partner and localizer of BRCA2 (PALB2), plays an important functional role in DNA damage repair. Recent studies indicate that germline mutations in PALB2 predispose individuals to a high risk of...
5.
Mdzinarishvili T, Sherman A, Shats O, Sherman S
Cancer Inform
. 2015 Mar;
13:197-205.
PMID: 25788828
A computational approach for estimating the overall, population, and individual cancer hazard rates was developed. The population rates characterize a risk of getting cancer of a specific site/type, occurring within...
6.
Wen H, Kim Y, Snyder C, Xiao F, Fleissner E, Becirovic D, et al.
BMC Cancer
. 2014 Jun;
14:470.
PMID: 24969172
Background: Genetic predisposition is the primary risk factor for familial breast cancer. For the majority of familial breast cancer, however, the genetic predispositions remain unknown. All newly identified predispositions occur...
7.
Mdzinarishvili T, Sherman S
PLoS One
. 2014 Jun;
9(6):e100087.
PMID: 24932779
At present, carcinogenic models imply that all individuals in a population are susceptible to cancer. These models either ignore a fall of the cancer incidence rate at old ages, or...
8.
Cheng P, Kumar V, Liu H, Youn J, Fishman M, Sherman S, et al.
Cancer Res
. 2013 Nov;
74(1):141-52.
PMID: 24220241
Functionally altered myeloid cells play an important role in immune suppression in cancer, in angiogenesis, and in tumor cells' invasion and metastases. Here, we report that inhibition of Notch signaling...
9.
Gleason M, Mdzinarishvili T, Are C, Sasson A, Sherman A, Shats O, et al.
Cancer Inform
. 2013 Apr;
12:103-14.
PMID: 23589669
The 18,352 pancreatic ductal adenocarcinoma (PDAC) cases from the Surveillance Epidemiology and End Results (SEER) database were analyzed using the Kaplan-Meier method for the following variables: race, gender, marital status,...
10.
Mdzinarishvili T, Sherman S
Cancer Inform
. 2013 Mar;
12:67-81.
PMID: 23471174
Modeling of cancer hazards at age t deals with a dichotomous population, a small part of which (the fraction at risk) will get cancer, while the other part will not....