Sherry M Mabry
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Explore the profile of Sherry M Mabry including associated specialties, affiliations and a list of published articles.
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19
Citations
239
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Recent Articles
1.
Xia S, Menden H, Mabry S, Sampath V
Cells
. 2023 Sep;
12(18).
PMID: 37759454
Angiogenesis plays a critical role in various physiological and pathological processes and is regulated by VEGF. Histone Deacetylase 6 (HDAC6) is a class IIB HDAC that regulates cytoplasmic signaling through...
2.
Menden H, Mabry S, Venkatraman A, Xia S, DeFranco D, Yu W, et al.
Am J Physiol Lung Cell Mol Physiol
. 2023 Mar;
324(5):L722-L736.
PMID: 36976925
SARS-CoV-2 viremia is associated with increased acute lung injury (ALI) and mortality in children and adults. The mechanisms by which viral components in the circulation mediate ALI in COVID-19 remain...
3.
Xia S, Ellis L, Winkley K, Menden H, Mabry S, Venkatraman A, et al.
Am J Physiol Lung Cell Mol Physiol
. 2022 Dec;
324(2):L123-L140.
PMID: 36537711
Hyperoxia disrupts lung development in mice and causes bronchopulmonary dysplasia (BPD) in neonates. To investigate sex-dependent molecular and cellular programming involved in hyperoxia, we surveyed the mouse lung using single...
4.
Yu W, Haque I, Venkatraman A, Menden H, Mabry S, Roy B, et al.
Cell Mol Gastroenterol Hepatol
. 2021 Sep;
13(2):425-440.
PMID: 34563711
Background & Aims: Single immunoglobulin interleukin-1-related receptor (SIGIRR) is a major inhibitor of Toll-like receptor signaling. Our laboratory identified a novel SIGIRR stop mutation (p.Y168X) in an infant who died...
5.
Salimi U, Menden H, Mabry S, Xia S, Sampath V
Pediatr Res
. 2021 May;
91(6):1405-1415.
PMID: 33980990
Background: Sepsis in premature newborns is a risk factor for bronchopulmonary dysplasia (BPD), but underlying mechanisms of lung injury remain unclear. Aberrant expression of endothelial cell (EC) angiopoietin 2 (ANGPT2)...
6.
Xia S, Menden H, Townley N, Mabry S, Johnston J, Nyp M, et al.
JCI Insight
. 2021 Apr;
6(7).
PMID: 33830085
The molecular mechanisms by which endothelial cells (ECs) regulate pulmonary vascularization and contribute to alveolar epithelial cell development during lung morphogenesis remain unknown. We tested the hypothesis that delta-like 4...
7.
Menden H, Xia S, Mabry S, Noel-MacDonnell J, Rajasingh J, Qing Ye S, et al.
Am J Physiol Lung Cell Mol Physiol
. 2019 Jul;
317(3):L332-L346.
PMID: 31268348
Lung endothelial cell (EC) immune activation during bacterial sepsis contributes to acute lung injury and bronchopulmonary dysplasia in premature infants. The epigenetic regulators of sepsis-induced endothelial immune activation, lung inflammation,...
8.
Nyp M, Mabry S, Navarro A, Menden H, Perez R, Sampath V, et al.
Physiol Rep
. 2018 Feb;
6(5).
PMID: 29484847
The onset and degree of injury occurring in animals that develop hyperoxic acute lung injury (HALI) is dependent on age at exposure, suggesting that developmentally regulated pathways/factors must underlie initiation...
9.
Menden H, Xia S, Mabry S, Navarro A, Nyp M, Sampath V
Am J Respir Cell Mol Biol
. 2016 Jul;
55(6):767-778.
PMID: 27438994
In premature infants, sepsis is associated with alveolar simplification manifesting as bronchopulmonary dysplasia. The redox-dependent mechanisms underlying sepsis-induced inflammation and alveolar remodeling in the immature lung remain unclear. We developed...
10.
Nyp M, Navarro A, Rezaiekhaligh M, Perez R, Mabry S, Ekekezie I
Respir Res
. 2014 Feb;
15:19.
PMID: 24528651
Background: Myofibroblasts are the critical effector cells in the pathogenesis of pulmonary fibrosis which carries a high degree of morbidity and mortality. We have previously identified Type II TGFβ receptor...