Sarah J Bartulis
Overview
Explore the profile of Sarah J Bartulis including associated specialties, affiliations and a list of published articles.
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Articles
5
Citations
151
Followers
0
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Recent Articles
1.
Murphy S, Rhee L, Grimm W, Weber C, Messer J, Lodolce J, et al.
Am J Physiol Gastrointest Liver Physiol
. 2014 Sep;
307(9):G871-82.
PMID: 25234043
Tumor necrosis factor-induced protein 3 (TNFAIP3; also known as A20) negatively regulates NF-κB and MAPK signals to control inflammatory responses. TNFAIP3 also protects against TNF-induced cell death. Intestinal epithelial cell...
2.
Messer J, Murphy S, Logsdon M, Lodolce J, Grimm W, Bartulis S, et al.
BMJ Open
. 2013 Jun;
3(6).
PMID: 23794574
Objective: A common genetic coding variant in the core autophagy gene ATG16L1 is associated with increased susceptibility to Crohn's disease (CD). The variant encodes an amino acid change in ATG16L1...
3.
Expression of TNFAIP3 in intestinal epithelial cells protects from DSS- but not TNBS-induced colitis
Rhee L, Murphy S, Kolodziej L, Grimm W, Weber C, Lodolce J, et al.
Am J Physiol Gastrointest Liver Physiol
. 2012 May;
303(2):G220-7.
PMID: 22595989
Intestinal epithelial cells (IEC) maintain gastrointestinal homeostasis by providing a physical and functional barrier between the intestinal lumen and underlying mucosal immune system. The activation of NF-κB and prevention of...
4.
Kolodziej L, Lodolce J, Chang J, Schneider J, Grimm W, Bartulis S, et al.
PLoS One
. 2011 Oct;
6(10):e26352.
PMID: 22031828
Tight junctions between intestinal epithelial cells mediate the permeability of the intestinal barrier, and loss of intestinal barrier function mediated by TNF signaling is associated with the inflammatory pathophysiology observed...
5.
Lodolce J, Kolodziej L, Rhee L, Kariuki S, Franek B, McGreal N, et al.
J Immunol
. 2010 May;
184(12):7001-9.
PMID: 20483768
The TNF alpha-induced protein 3 (TNFAIP3) is an ubiquitin-modifying enzyme and an essential negative regulator of inflammation. Genome-wide association studies have implicated the TNFAIP3 locus in susceptibility to autoimmune disorders...