Samuel Shribman
Overview
Explore the profile of Samuel Shribman including associated specialties, affiliations and a list of published articles.
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Articles
16
Citations
348
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0
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Recent Articles
1.
Shribman S, Lachmann R
Pract Neurol
. 2024 Jul;
24(5):354-356.
PMID: 38960598
No abstract available.
2.
Pereira A, Alakbarzade V, Shribman S, Crossingham G, Moullaali T, Werring D
Pract Neurol
. 2024 Jun;
25(1):45-50.
PMID: 38908861
Stroke is one of the most common acute neurological disorders and a leading cause of disability worldwide. Evidence-based treatments over the last two decades have driven a revolution in the...
3.
Cullinane P, Wrigley S, Bradshaw T, Shaw K, Shribman S, De Pablo Fernandez E, et al.
Mov Disord Clin Pract
. 2023 Sep;
10(9):1414-1418.
PMID: 37772307
Background: Chronic traumatic encephalopathy (CTE) is a neurodegenerative disease characterized by widespread accumulation of hyperphosphorylated tau that typically occurs in people who have suffered repetitive head impacts. To date, very...
4.
Shribman S, Burrows M, Convery R, Bocchetta M, Sudre C, Acosta-Cabronero J, et al.
Mov Disord
. 2022 Jun;
37(8):1728-1738.
PMID: 35723521
Background: Cognitive impairment is common in neurological presentations of Wilson's disease (WD). Various domains can be affected, and subclinical deficits have been reported in patients with hepatic presentations. Associations with...
5.
Shribman S, Marjot T, Sharif A, Vimalesvaran S, Ala A, Alexander G, et al.
Lancet Gastroenterol Hepatol
. 2022 Apr;
7(6):560-575.
PMID: 35429442
Wilson's disease is an autosomal-recessive disorder of copper metabolism with hepatic, neurological, psychiatric, ophthalmological, haematological, renal, and rheumatological manifestations. Making a diagnosis can be challenging given that no single test...
6.
Shribman S, Poujois A, Bandmann O, Czlonkowska A, Warner T
J Neurol Neurosurg Psychiatry
. 2021 Aug;
92(10):1053-1061.
PMID: 34341141
Wilson's disease is an autosomal-recessive disorder of copper metabolism caused by mutations in and associated with neurological, psychiatric, ophthalmological and hepatic manifestations. Decoppering treatments are used to prevent disease progression...
7.
Shribman S, Bocchetta M, Sudre C, Acosta-Cabronero J, Burrows M, Cook P, et al.
Brain
. 2021 Jul;
145(1):263-275.
PMID: 34289020
Wilson's disease is an autosomal-recessive disorder of copper metabolism with neurological and hepatic presentations. Chelation therapy is used to 'de-copper' patients but neurological outcomes remain unpredictable. A range of neuroimaging...
8.
Shribman S, Heller C, Burrows M, Heslegrave A, Swift I, Foiani M, et al.
Mov Disord
. 2020 Oct;
36(2):503-508.
PMID: 33078859
Background: Outcomes are unpredictable for neurological presentations of Wilson's disease (WD). Dosing regimens for chelation therapy vary and monitoring depends on copper indices, which do not reflect end-organ damage. Objective:...
9.
Shribman S, Webb G, Taylor R, Warner T, Duckworth A, Gimson A, et al.
JHEP Rep
. 2020 Apr;
2(3):100096.
PMID: 32322813
Background & Aims: Acute liver failure as the initial presentation of Wilson's disease is usually associated with onset in childhood, adolescence or early adulthood. Outcomes after transplantation for late-onset presentations,...
10.
Shribman S, Reid E, Crosby A, Houlden H, Warner T
Lancet Neurol
. 2019 Aug;
18(12):1136-1146.
PMID: 31377012
Hereditary spastic paraplegia (HSP) describes a heterogeneous group of genetic neurodegenerative diseases characterised by progressive spasticity of the lower limbs. The pathogenic mechanism, associated clinical features, and imaging abnormalities vary...