Sameer Agnihotri
Overview
Explore the profile of Sameer Agnihotri including associated specialties, affiliations and a list of published articles.
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Articles
107
Citations
4174
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Recent Articles
1.
Xiong Z, Walsh K, Sneiderman C, Nisnboym M, Hadjipanayis C, Agnihotri S, et al.
Neuro Oncol
. 2025 Feb;
PMID: 39949048
Background: Individual-level characteristics underlying population-level variation in glioma risk and outcomes remain incompletely understood. Cancer immunosurveillance, host immunity, and some immunotherapies center on the ability of an individual's immune cells...
2.
Gao J, Gu D, Yang K, Zhang J, Lin Q, Yuan W, et al.
Cancer Cell
. 2025 Jan;
43(1):122-143.e8.
PMID: 39753140
Glioblastoma is a highly aggressive primary brain tumor with glioblastoma stem cells (GSCs) enforcing the intra-tumoral hierarchy. Plasma cells (PCs) are critical effectors of the B-lineage immune system, but their...
3.
Lv D, Dixit D, Cruz A, Kim L, Duan L, Xu X, et al.
Cell Metab
. 2024 Oct;
36(11):2419-2436.e8.
PMID: 39454581
Tumors reprogram their metabolism to generate complex neoplastic ecosystems. Here, we demonstrate that glioblastoma (GBM) stem cells (GSCs) display elevated activity of the malate-aspartate shuttle (MAS) and expression of malate...
4.
Mbah N, Myers A, Sajjakulnukit P, Chung C, Thompson J, Hong H, et al.
Nat Commun
. 2024 Oct;
15(1):8983.
PMID: 39419964
H3K27M diffuse midline gliomas (DMG), including diffuse intrinsic pontine gliomas (DIPG), exhibit cellular heterogeneity comprising less-differentiated oligodendrocyte precursors (OPC)-like stem cells and more differentiated astrocyte (AC)-like cells. Here, we establish...
5.
Nisnboym M, Sneiderman C, Jaswal A, Xiong Z, Vincze S, Sever R, et al.
Expert Rev Clin Immunol
. 2024 Oct;
21(2):239-247.
PMID: 39402706
Background: Glioblastoma (GBM) is an aggressive cancer with limited treatment options. Immunotherapy targeting CD69, an early activation marker on T cells, has shown promise in preclinical models of non-CNS malignancies....
6.
Guo Y, Li Z, Parsels L, Wang Z, Parsels J, Dalvi A, et al.
bioRxiv
. 2024 Sep;
PMID: 39253432
Background: Radiotherapy (RT) is the primary treatment for diffuse midline glioma (DMG), a lethal pediatric malignancy defined by histone H3 lysine 27-to-methionine (H3K27M) mutation. Based on the loss of H3K27...
7.
Andrade A, Annett A, Karimi E, Topouza D, Rezanejad M, Liu Y, et al.
Nat Commun
. 2024 Sep;
15(1):7769.
PMID: 39237515
Histone H3-mutant gliomas are deadly brain tumors characterized by a dysregulated epigenome and stalled differentiation. In contrast to the extensive datasets available on tumor cells, limited information exists on their...
8.
Liu I, Cruzeiro G, Bjerke L, Rogers R, Grabovska Y, Beck A, et al.
Cancer Cell
. 2024 Sep;
PMID: 39232581
Diffuse hemispheric gliomas, H3G34R/V-mutant (DHG-H3G34), are lethal brain tumors lacking targeted therapies. They originate from interneuronal precursors; however, leveraging this origin for therapeutic insights remains unexplored. Here, we delineate a...
9.
Johnston M, Lee J, Hu B, Nikolic A, Hasheminasabgorji E, Baguette A, et al.
Cell
. 2024 Jul;
187(18):4926-4945.e22.
PMID: 38986619
Posterior fossa group A (PFA) ependymoma is a lethal brain cancer diagnosed in infants and young children. The lack of driver events in the PFA linear genome led us to...
10.
Rao A, Zhang X, Cillo A, Sussman J, Sandlesh P, Tarbay A, et al.
bioRxiv
. 2024 Apr;
PMID: 38645178
Diffuse gliomas are epigenetically dysregulated, immunologically cold, and fatal tumors characterized by mutations in isocitrate dehydrogenase (IDH). Although IDH mutations yield a uniquely immunosuppressive tumor microenvironment, the regulatory mechanisms that...