» Authors » S S Brooks

S S Brooks

Explore the profile of S S Brooks including associated specialties, affiliations and a list of published articles. Areas
Snapshot
Articles 17
Citations 171
Followers 0
Related Specialties
Top 10 Co-Authors
Published In
Affiliations
Soon will be listed here.
Recent Articles
1.
Berliner J, Shapiro F, Nolin S, Houck Jr G, Ding X, Dobkin C, et al.
J Genet Couns . 2013 Nov; 3(3):233-44. PMID: 24234009
Molecular analysis of the fragile X (FMR-1) gene identifies female fragile X carriers, but appropriate genetic counseling can only be provided if the limitations of the testing methods are understood....
2.
Ashkinadze E, Rosen T, Brooks S, Katsanis N, Davis E
Clin Genet . 2012 Sep; 83(6):553-9. PMID: 22998390
Bardet-Biedl syndrome (BBS) is a rare pediatric ciliopathy characterized by marked clinical variability and extensive genetic heterogeneity. Typical diagnosis of BBS is secured at a median of 9 years of...
3.
Puvabanditsin S, Garrow E, Lambert G, Botti C, Gowda S, Kathiravan S, et al.
Genet Couns . 2011 Oct; 22(3):263-72. PMID: 22029167
We report on a preterm neonate with a deletion of the distal long arm of chromosome 13q32.1 and partial trisomy of the short arm of chromosome 10p12.33. The patient has...
4.
Junaid M, Brooks S, Pullarkat R
Eur J Paediatr Neurol . 2001 Oct; 5 Suppl A:63-8. PMID: 11589010
The classic late infantile neuronal ceroid lipofuscinosis (LINCL, CLN2) is a fatal neurodegenerative disorder that results from mutations in a gene encoding a lysosomal proteinase, known as CLN2 protease (CLN2p)...
5.
Combes A, McTiernan C, Brooks S, Feldman A
J Card Fail . 2001 Jun; 7(2):165-75. PMID: 11420769
Background: Proinflammatory cytokines play an important role in chronic cardiac diseases. Methods And Results: Neonatal rat cardiomyocytes were exposed to interleukin (IL)-1beta (2 ng/mL) for 4 days. We assessed contractility...
6.
Brooks S
Adv Genet . 2001 May; 45:159-67. PMID: 11332771
No abstract available.
7.
Zhang Z, Butler J, Levin S, Wisniewski K, Brooks S, Mukherjee A
Nat Med . 2001 Apr; 7(4):478-84. PMID: 11283676
Neuronal ceroid lipofuscinoses (NCLs) are the most common hereditary neurodegenerative diseases of childhood. The infantile form, INCL, is caused by lysosomal palmitoyl-protein thioesterase (PPT) deficiency, which impairs the cleavage of...
8.
Becker J, Vlach J, Raben N, Nagaraju K, Adams E, Hermans M, et al.
Am J Hum Genet . 1998 Jun; 62(4):991-4. PMID: 9529346
No abstract available.
9.
Wisniewski K, Zhong N, Kida E, Kaczmarski W, Kaczmarski A, Connell F, et al.
Folia Neuropathol . 1997 Jan; 35(2):73-9. PMID: 9377079
We have collected 122 late-infantile neuronal ceroid lipofuscinosis (LINCL, CLN2) and 191 juvenile NCL (JNCL, CLN3) cases, diagnosed on the basis of age-at-onset, clinical symptomatology, and pathological findings and representing...
10.
Giampietro P, Haas B, Lipper E, Gutman A, Zellers N, Latrenta G, et al.
Am J Med Genet . 1996 May; 63(2):396-400. PMID: 8725793
We report on 2 brothers with both fragile X and VACTERL-H syndrome. The first sibling, age 5, had bilateral cleft lip and palate, ventricular septal defect, and a hypoplastic thumb....