Rongheng Liao
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Explore the profile of Rongheng Liao including associated specialties, affiliations and a list of published articles.
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7
Citations
50
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0
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Recent Articles
1.
Zhang L, van Schie M, Xiang H, Liao R, Zheng J, Knops P, et al.
J Clin Med
. 2024 Feb;
13(4).
PMID: 38398329
(1) Background: Structural remodeling plays an important role in the pathophysiology of atrial fibrillation (AF). It is likely that structural remodeling occurs transmurally, giving rise to electrical endo-epicardial asynchrony (EEA)....
2.
van Schie M, Liao R, Ramdat Misier N, Knops P, Heida A, Taverne Y, et al.
Europace
. 2023 Jul;
25(9).
PMID: 37477953
Background And Aims: Atrial extrasystoles (AES) provoke conduction disorders and may trigger episodes of atrial fibrillation (AF). However, the direction- and rate-dependency of electrophysiological tissue properties on epicardial unipolar electrogram...
3.
Qi Z, Wang R, Liao R, Xue S, Wang Y
Front Pharmacol
. 2022 May;
13:913778.
PMID: 35571110
[This corrects the article DOI: 10.3389/fphar.2021.706251.].
4.
Liao R, Xie B, Cui J, Qi Z, Xue S, Wang Y
Bioengineered
. 2021 Sep;
12(1):6869-6877.
PMID: 34521301
The differentiation of cardiac fibroblast to myofibroblast is the key process of cardiac fibrosis. In the study, we aimed to determine the function of E2F Transcription Factor 1 (E2F1) in...
5.
Liao R, Qi Z, Tang R, Wang R, Wang Y
Front Pharmacol
. 2021 Sep;
12:714390.
PMID: 34483923
Myocardial fibrosis is a key pathological process after myocardial infarction, which leads to poor outcomes in patients at the end stage. Effective treatments for improving prognosis of myocardial fibrosis are...
6.
Qi Z, Wang R, Liao R, Xue S, Wang Y
Front Pharmacol
. 2021 Aug;
12:706251.
PMID: 34366860
Septic cardiomyopathy is a common complication of severe sepsis, which is one of the leading causes of death in intensive care units. Therefore, finding an effective therapy target is urgent....
7.
Cui J, Qi S, Liao R, Su D, Wang Y, Xue S
Biochem Biophys Res Commun
. 2019 Nov;
521(2):427-433.
PMID: 31672272
Cardiac fibrosis after myocardial infarction (MI) is mainly associated with cardiac fibroblasts and its differentiation is the key pathological process. However, the cellular mechanism of fibroblast-to-myofibroblast conversion has not been...