Ralph DeFronzo
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Explore the profile of Ralph DeFronzo including associated specialties, affiliations and a list of published articles.
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50
Citations
3627
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Recent Articles
1.
Baskoy G, Peterson R, Kempenich J, Triplitt C, Brown M, Clarke G, et al.
J Clin Endocrinol Metab
. 2024 Dec;
PMID: 39689013
Context: Increased mesenteric visceral fat is associated with the metabolic syndrome, insulin resistance, and type 2 diabetes. Methods: Using Targeted Cell Separation and Extraction Technology (TC-SET), we examined the effect...
2.
Rayas M, Pezzica S, Honka H, Carli F, Peterson R, DeFronzo R, et al.
Obesity (Silver Spring)
. 2024 Dec;
33(1):104-115.
PMID: 39635951
Objective: The glycemic-independent actions of glucagon-like peptide-1 (GLP-1) in the prandial state in humans are unknown. We examined the contribution of GLP-1 to β-cell secretory response (primary endpoint) and glucose...
3.
Honka H, Gastaldelli A, Pezzica S, Peterson R, DeFronzo R, Salehi M
Diabetes Obes Metab
. 2024 Apr;
26(6):2476-2486.
PMID: 38558527
Aim: To determine the effect of endogenous glucagon-like peptide 1 (GLP-1) on prandial counterregulatory response to hypoglycaemia after gastric bypass (GB). Materials And Methods: Glucose fluxes, and islet-cell and gut...
4.
Busch C, Meiring S, van Baar A, Gastaldelli A, DeFronzo R, Mingrone G, et al.
Gastrointest Endosc
. 2024 Jan;
100(3):473-480.e1.
PMID: 38280531
Background And Aims: The duodenum has been shown to play a key role in glucose homeostasis. Duodenal mucosal resurfacing (DMR) is an endoscopic procedure for patients with type 2 diabetes...
5.
Rayas M, Gastaldelli A, Honka H, Pezzica S, Carli F, Peterson R, et al.
medRxiv
. 2023 Nov;
PMID: 37961500
Objective: Protein ingestion stimulates β-cell secretion and alters glucose flux. Enhanced action of glucagon-like peptide 1 (GLP-1) and increased plasma glucose excursion contribute to prandial hyperinsulinemia after gastric bypass surgery...
6.
Salehi M, Peterson R, Tripathy D, Pezzica S, DeFronzo R, Gastaldelli A
Obesity (Silver Spring)
. 2023 Oct;
31(11):2774-2785.
PMID: 37853989
Objective: Prandial hyperinsulinemia after Roux-en-Y gastric bypass surgery (GB), and to lesser degree after sleeve gastrectomy (SG), has been attributed to rapid glucose flux from the gut and increased insulinotropic...
7.
Honka H, Gastaldelli A, Pezzica S, Peterson R, DeFronzo R, Salehi M
medRxiv
. 2023 Oct;
PMID: 37790563
We have previously shown that prandial endogenous glucose production (EGP) during insulin-induced hypoglycemia is smaller in non-diabetic subjects with gastric bypass (GB), where prandial glucagon-like peptide 1 (GLP-1) concentrations are...
8.
Bariatric Surgery Alters the Postprandial Recovery From Hypoglycemia, Mediated by Cholinergic Signal
Salehi M, Tripathy D, Peterson R, Honka H, Pezzica S, DeFronzo R, et al.
Diabetes
. 2023 Jul;
72(10):1374-1383.
PMID: 37467435
Article Highlights: Rerouted gut after Roux-en-Y gastric bypass (GB) and, to a lesser degree, after sleeve gastrectomy (SG) leads to larger glucose excursion and lower nadir glucose, predisposing individuals to...
9.
Salehi M, Peterson R, Tripathy D, Pezzica S, DeFronzo R, Gastaldelli A
medRxiv
. 2023 Apr;
PMID: 37034666
Background/aims: Prandial hyperinsulinemia after Roux-en Y gastric bypass surgery (GB), and to lesser degree after sleeve gastrectomy (SG), has been attributed to rapid glucose flux from the gut and increased...
10.
Salehi M, DeFronzo R, Gastaldelli A
Int J Mol Sci
. 2022 Jul;
23(14).
PMID: 35887007
Background: The liver has the capacity to regulate glucose metabolism by altering the insulin clearance rate (ICR). The decreased fasting insulin concentrations and enhanced prandial hyperinsulinemia after Roux-en-Y gastric-bypass (GB)...