Raimund Trabold
Overview
Explore the profile of Raimund Trabold including associated specialties, affiliations and a list of published articles.
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Articles
24
Citations
555
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0
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Recent Articles
1.
Schmutzer-Sondergeld M, Zimmermann H, Trabold R, Liebig T, Schichor C, Siller S
Acta Neurochir (Wien)
. 2023 Sep;
165(11):3479-3491.
PMID: 37743433
Background: In contrast to osteoligamentous lumbar stenosis (LSS), outcome of surgical treatment for spinal epidural lipomatosis (SEL) is still not well defined. We present risk factors for SEL and clinical...
2.
Siller S, Skrap B, Grabein B, Trabold R, Zausinger S, Tonn J
Acta Neurochir (Wien)
. 2022 Jan;
164(3):891-901.
PMID: 35080653
Purpose: Surgical site infections (SSI) are a rare but dreaded cause for recurrent symptomatology requiring reoperation after degenerative spine surgery. We here aim to elucidate if routine microbiological smear testing...
3.
Siller S, Zoellner C, Fuetsch M, Trabold R, Tonn J, Zausinger S
J Neurosurg Spine
. 2020 Jul;
33(5):705-714.
PMID: 32650307
Objective: Since the 1970s, the operating microscope (OM) has been a standard for visualization and illumination of the surgical field in spinal microsurgery. However, due to its limitations (e.g., size,...
4.
Rauen K, Pop V, Trabold R, Badaut J, Plesnila N
J Neurotrauma
. 2019 Sep;
37(4):665-674.
PMID: 31547764
Brain edema formation contributes to secondary brain damage and unfavorable outcome after traumatic brain injury (TBI). Aquaporins (AQP), highly selective water channels, are involved in the formation of post-trauma brain...
5.
Griemert E, Schwarzmaier S, Hummel R, Golz C, Yang D, Neuhaus W, et al.
Ann Neurol
. 2019 Mar;
85(5):667-680.
PMID: 30843275
Objective: Plasminogen activator inhibitor-1 (PAI-1) is the key endogenous inhibitor of fibrinolysis, and enhances clot formation after injury. In traumatic brain injury, dysregulation of fibrinolysis may lead to sustained microthrombosis...
6.
Krieg S, Trabold R, Plesnila N
J Neurotrauma
. 2016 Oct;
34(7):1329-1336.
PMID: 27762660
Arginine-vasopressin (AVP) V receptors are known to mediate brain edema formation after traumatic brain injury (TBI). So far, however, AVP V receptors were only inhibited by genetic deletion or prior...
7.
Schniepp R, Trabold R, Romagna A, Akrami F, Hesselbarth K, Wuehr M, et al.
J Neurosurg
. 2016 Mar;
126(1):148-157.
PMID: 26991388
OBJECTIVE The determination of gait improvement after lumbar puncture (LP) in idiopathic normal-pressure hydrocephalus (iNPH) is crucial, but the best time for such an assessment is unclear. The authors determined...
8.
Krieg S, Sonanini S, Plesnila N, Trabold R
J Neurotrauma
. 2014 Aug;
32(4):221-7.
PMID: 25111427
The attenuation of brain edema is a major therapeutic target after traumatic brain injury (TBI). Vasopressin (AVP) is well known to play a major role in the regulation of brain...
9.
Eigenbrod S, Trabold R, Brucker D, Eros C, Egensperger R, Fougere C, et al.
Acta Neurochir (Wien)
. 2014 May;
156(8):1427-40.
PMID: 24792966
Background: In gliomas molecular biomarkers are increasingly gaining diagnostic, prognostic and predictive significance. Determination of biomarker status after biopsy is important as not all patients are eligible for open tumor...
10.
Schwarzmaier S, Zimmermann R, McGarry N, Trabold R, Kim S, Plesnila N
J Neuroinflammation
. 2013 Mar;
10:32.
PMID: 23448240
Background: Leukocytes are believed to be involved in delayed cell death following traumatic brain injury (TBI). However, data demonstrating that blood-borne inflammatory cells are present in the injured brain prior...