Philippe Chadebech
Overview
Explore the profile of Philippe Chadebech including associated specialties, affiliations and a list of published articles.
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Articles
19
Citations
163
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Recent Articles
1.
Bencheikh L, Nguyen K, Chadebech P, Kiger L, Bodivit G, Jouard A, et al.
Haematologica
. 2022 Mar;
107(8):1944-1949.
PMID: 35354249
No abstract available.
2.
Chadebech P, Bodivit G, Di Liberto G, Jouard A, Vasseur C, Pirenne F, et al.
Biomolecules
. 2021 Jul;
11(7).
PMID: 34208829
Background: Blood transfusion remains a key treatment for managing occlusive episodes and painful crises in sickle-cell disease (SCD). In that clinical context, red blood cells (RBCs) from donors and transfused...
3.
Domingues-Hamdi E, Vasseur C, Bodivit G, Jouard A, de Menorval M, Pirenne F, et al.
Blood Transfus
. 2021 Feb;
20(2):127-131.
PMID: 33539282
No abstract available.
4.
Le Toriellec E, Muralitharan V, Chadebech P, Jouard A, Ansart-Pirenne H, Pirenne F, et al.
Transfusion
. 2020 Sep;
60(11):2482-2488.
PMID: 32949421
Background: CD36 glycoprotein is expressed by various cell types, including platelets (PLTs), monocytes, and erythroid precursors, and is also the receptor for several ligands. However, absence of CD36 expression seems...
5.
Chadebech P, de Menorval M, Bodivit G, Jouard A, Pakdaman S, Lelong F, et al.
Cytokine
. 2020 Sep;
136:155259.
PMID: 32920319
Background: Changes in cytokine production are known to contribute to the pathogenesis of sickle-cell disease (SCD), particularly in painful acute complications (crises) and episodes of post-transfusion hemolysis. Little is known...
6.
Hebert N, Rakotoson M, Bodivit G, Audureau E, Bencheikh L, Kiger L, et al.
Am J Hematol
. 2020 Jul;
95(11):1235-1245.
PMID: 32681733
Polymerization of the sickle hemoglobin (HbS) is a key determinant of sickle cell disease (SCD), an inherited blood disorder. Fetal hemoglobin (HbF) is a major modulator of the disease severity...
7.
Roumenina L, Chadebech P, Bodivit G, Vieira-Martins P, Grunenwald A, Boudhabhay I, et al.
Am J Hematol
. 2020 Jan;
95(5):456-464.
PMID: 31990387
The complement system is an innate immune defense cascade that can cause tissue damage when inappropriately activated. Evidence for complement over activation has been reported in small cohorts of patients...
8.
Burin des Roziers N, Chadebech P, Malard L, Vingert B, Gallon P, Samuel D, et al.
Transfusion
. 2019 Jan;
59(4):1353-1358.
PMID: 30604873
Background: Recent reports have indicated that the risk of anti-D alloimmunization following D-incompatible platelet (PLT) transfusion is low in hematology and oncology patients. We investigated the rate of anti-D alloimmunization...
9.
Chadebech P, Loustau V, Janvier D, Languille L, Ripa J, Tamagne M, et al.
Haematologica
. 2017 Oct;
103(1):e35-e38.
PMID: 29025905
No abstract available.
10.
Narbey D, Habibi A, Chadebech P, Mekontso-Dessap A, Khellaf M, Lelievre J, et al.
Am J Hematol
. 2017 Sep;
92(12):1340-1348.
PMID: 28924974
Delayed hemolytic transfusion reaction (DHTR) is a life-threatening complication of transfusion in sickle cell disease (SCD). The frequency of DHTR is underestimated because its symptoms mimic those of vaso-occlusive crisis...