Paulo D Koeberle
Overview
Explore the profile of Paulo D Koeberle including associated specialties, affiliations and a list of published articles.
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Articles
19
Citations
476
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0
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Recent Articles
1.
Shabanzadeh A, Charish J, Tassew N, Farhani N, Feng J, Qin X, et al.
Neurobiol Dis
. 2021 Jan;
150:105259.
PMID: 33434618
Neuronal regeneration in the injured central nervous system is hampered by multiple extracellular proteins. These proteins exert their inhibitory action through interactions with receptors that are located in cholesterol rich...
2.
Choi B, DOnofrio P, Shabanzadeh A, Koeberle P
PLoS One
. 2019 Aug;
14(8):e0220056.
PMID: 31369591
Abortive cell cycle (ACC) re-entry of apoptotic neurons is a recently characterized phenomenon that occurs after central nervous system (CNS) injury or over the course of CNS disease. Consequently, inhibiting...
3.
DOnofrio P, Shabanzadeh A, Choi B, Bahr M, Koeberle P
Invest Ophthalmol Vis Sci
. 2019 Feb;
60(2):634-649.
PMID: 30743263
Purpose: Integrin adherence to the extracellular matrix (ECM) is essential for retinal ganglion cell (RGC) survival: damage causes production and release of ECM degrading matrix metalloproteinases (MMPs) that disrupt integrin...
4.
Shabanzadeh A, DOnofrio P, Monnier P, Koeberle P
J Stroke Cerebrovasc Dis
. 2017 Dec;
27(4):845-856.
PMID: 29196198
Background: A reliable model of ischemia-reperfusion is required to evaluate the efficacy and safety of neuroprotective therapies for stroke. We present a novel reproducible pterygopalatine-ophthalmic artery ligation model of ischemia-reperfusion...
5.
Tassew N, Charish J, Shabanzadeh A, Luga V, Harada H, Farhani N, et al.
Cell Rep
. 2017 Jul;
20(1):99-111.
PMID: 28683327
Developing strategies that promote axonal regeneration within the injured CNS is a major therapeutic challenge, as axonal outgrowth is potently inhibited by myelin and the glial scar. Although regeneration can...
6.
Tassew N, Mothe A, Shabanzadeh A, Banerjee P, Koeberle P, Bremner R, et al.
Cell Rep
. 2014 Aug;
8(4):1146-59.
PMID: 25127134
Ideal strategies to ameliorate CNS damage should promote both neuronal survival and axon regeneration. The receptor Neogenin promotes neuronal apoptosis. Its ligand prevents death, but the resulting repulsive guidance molecule...
7.
Galan A, Dergham P, Escoll P, de-la-Hera A, DOnofrio P, Magharious M, et al.
PLoS One
. 2014 Jul;
9(7):e101349.
PMID: 24983470
Retinal ganglion cells (RGCs) are neurons that relay visual signals from the retina to the brain. The RGC cell bodies reside in the retina and their fibers form the optic...
8.
Siddiqui A, Sabljic T, Koeberle P, Ball A
Invest Ophthalmol Vis Sci
. 2014 Feb;
55(3):1919-29.
PMID: 24526440
Purpose: BM88 is a cell-cycle exit and neuronal differentiation protein that has been used as a marker of surviving retinal ganglion cells (RGCs) after optic nerve injury. Thy1.1 has also...
9.
DOnofrio P, Koeberle P
Acta Pharmacol Sin
. 2012 Dec;
34(1):91-103.
PMID: 23202803
Retinal ischemia is a very useful model to study the impact of various cell death pathways, such as apoptosis and necrosis, in the ischemic retina. However, it is important to...
10.
Hollander A, DOnofrio P, Magharious M, Lysko M, Koeberle P
Invest Ophthalmol Vis Sci
. 2012 Apr;
53(7):3973-89.
PMID: 22531700
Purpose: Retinal ganglion cell (RGC) degeneration is an important cause of visual impairment and can be modeled by optic nerve transection, which causes the death of 90% of RGCs within...